Acute myocardial infarction is caused by acute narrowing or occlusion of the coronary artery lumen as a result of thrombosis induced by rupture of atherosclerotic plaques in the coronary arteries. The most important cause of death in acute myocardial infarction is cardiac arrest due to ventricular fibrillation. 3 to 5 minutes after acute myocardial infarction occurs, the myocardium in the infarcted area will soon lose its contractility and produce reduced or absent left heart systolic function, which can be clinically manifested as varying degrees of left heart failure, pulmonary edema, and even cardiogenic shock. Due to the occurrence of cardiogenic shock, the systemic tissues and blood pressure become hypoperfused, and death occurs due to the inability to perform normal metabolic activities. There are also a few cases of pericardial compression due to rupture of the free wall of the heart. Therefore, the process of death due to acute myocardial infarction is diverse and should be promptly identified and actively resuscitated.