High duodenal dilatation is one of the clinical symptoms of acute jejunal input collaterals obstruction in gastrojejunal input collaterals syndrome and is one of the causes of acute gastric dilatation. So how does high duodenal dilatation occur? Here is a brief introduction for you. 1, gastric and intestinal wall neuromuscular paralysis Due to injury to our visceral nerves, as well as the stretching of our abdomen by surgery, etc., can lead to a high degree of dilatation of our duodenum. Large amounts of food can cause reflex paralysis of the stomach wall muscles due to sudden overstretching. When the stomach and duodenum are paralyzed. Dilatation occurs due to its reduced muscle tone. Gastric juice, bile, pancreatic residue and duodenal fluid secreted from the upper gastrointestinal tract, together with food fermentation and swallowed gas, can cause further expansion of the stomach and duodenum, forming a vicious circle in a short time, thus triggering a high degree of duodenal dilatation. 2, various traumatic injuries produced by the stress state Especially upper abdominal contusions or serious compound injuries, the occurrence of which is related to the strong stimulation of the abdominal plexus. Overeating can lead to reflex paralysis triggered by excessive stretching of the stomach wall muscles, which can also produce a high degree of duodenal dilatation. When the stomach dilates to a certain degree, the muscle tone of the stomach wall weakens, causing an acute angle to form at the junction of the esophagus and cardia, stomach and duodenum, which hinders the discharge of gastric contents. The enlarged stomach can compress the duodenum and squeeze the mesentery and small intestine into the pelvis, resulting in compression of the duodenum by the superior mesenteric artery and causing obstruction of the distal pylorus. Excessive secretion of saliva and gastric and duodenal fluids or pancreatic juice can aggravate the dilatation of the intestine and stomach, and also compress our portal vein. It can also compress the inferior vena cava, reducing the amount of blood returned to the heart and eventually leading to peripheral circulation failure. Due to massive vomiting, fasting and gastrointestinal decompression and drainage, it can cause water and electrolyte disorders.