The thinning of the renal cortex often suggests a state of high pressure within the renal pelvis or an ischemic state and other factors, resulting in progressive damage to the glomerular renal unit before the renal cortex becomes thin. Treatment: 1. Improve relevant examinations to clarify the presence of obstructive factors, such as ureteral tumors, ureteral stones, pelvic ureteral junction stenosis, prostatic hyperplasia, long-term urinary retention, etc. After the cause of the obstruction is clearly identified, surgery is needed to remove the obstruction to reduce the pressure in the renal pelvis and avoid further cortical thinning of the kidney, such as pyeloplasty, ureteroscopic lithotomy or electrodesiccation of the prostate to relieve the obstruction and reduce the pressure in the renal pelvis. 2. Various occupational or ischemic diseases, such as tumors in the kidney or multiple cysts in the kidney, cause cortical compression and thinning. thinning. The cause should be clarified and the primary disease should be removed, such as radical surgery for kidney tumor or cyst puncture, cyst decompression surgery, etc., to reduce the compression factors in the kidney. 3. Improve the blood supply, such as the presence of tumor embolism or vascular embolism in the blood vessels that cause local infarction in the kidney, or ischemia, etc., resulting in kidney atrophy and cortical thinning. It is necessary to perform renal arteriography to clarify the cause of insufficient blood supply and to perform comprehensive treatment such as stent expansion.