Sensory, muscular, and neurological abnormalities of the lower extremities often occur in thoracic spinal stenosis, and the clinical manifestations of the disease are mainly a series of syndromes of blood supply circulation to the thoracic spinal cord, sensory, muscular, and neurological abnormalities of the lower extremities caused by incomplete compression of the thoracic medulla. Thoracic spinal stenosis is a disease in which the thoracic spinal cord and nerve roots are compressed due to congenital or acquired degenerative factors, resulting in corresponding clinical symptoms and signs. The thoracic segment of the spinal cord has impaired blood supply and circulation, sensory and motor conduction caused by the compressor. There are many mechanisms that cause thoracic spinal stenosis, including congenital: spinal canal hypoplasia and shortened pedicles; hereditary abnormalities of bone metabolism such as Paget’s disease; and Vit-D resistant bone disease. There are also acquired ones: nephrotic abnormalities of bone metabolism, fluorosis. The most common clinical condition is due to strain factors. 1.Symptom examination Abnormal sensation of the lower limbs, such as numbness, dull sensation, feeling of cotton on the feet, appearance of sensory planes; abnormal muscle strength of the lower limbs, such as weakness, difficulty walking; increased muscle tone of the lower limbs, muscle tension, folding knife-like spasm; abnormal nerve reflexes, such as active or hyperactive knee and ankle reflexes, knee and ankle clonus, positive Barbinskin sign; nerve root irritation symptoms, such as thoracic dorsal fasciculation, pain The spinal cord, cauda equina circulation disorder, intermittent claudication of neurogenic origin; sphincter dysfunction, diaphoresis; complete spinal cord compression in advanced stage, paraplegia, diaphoresis incontinence, etc. 2, MRI examination of the spine MRI examination of the spine is the correct rate of diagnosis of spinal and spinal cord diseases MRI is significantly higher than CT, the source of disease display, accurate localization, can be the preferred method of examination. If pathological tissue is included, the order of decreasing brightness on T1-weighted imaging is fat, bone marrow, 4- to 5-day old hemorrhage, protein-rich fluid (e.g., necrotic tissue), mucus, melanin (melanin), slow blood flow (e.g., venous blood) free radicals, and GDDTPA (for MRI enhancer. The order of decreasing brightness in T2-weighted imaging is tumor, gliosis (gliosis), edema, peri-l stale hemorrhage, fluid, and vertebral interrogative disc. Those with dark (low) signal on both T1- and T2-weighted imaging are air, rapid blood flow (e.g., arterial blood), calcium, iron, fresh blood over several days, ligaments, tendons, and other magnetically sensitive material.