Right ventricular hypertrophy is usually the cause of pulmonary hypertension, and over time it can line up as right ventricular hypertrophy which is a compensatory condition that requires treatment of the primary disease. The following describes how to diagnose and treat patients with right ventricular hypertrophy? Diagnosis Right ventricular hypertrophy is a compensatory change in the heart. It is important to actively treat the primary disease to relieve the systolic or diastolic load of the right ventricle. Right heart hypertrophy can worsen and lead to right heart failure if the condition continues to worsen. A controlled electrocardiogram and echocardiogram were performed in patients with pulmonary heart disease to observe and compare the sensitivity of the two tests for the diagnosis of early right ventricular hypertrophy in pulmonary heart disease. Myocardial hypertrophy is usually seen on ECG or ultrasound, but may manifest itself as dyspnea, ventricular murmurs, etc. 1.Identify high-risk groups: Those with underlying diseases listed in the classification table are high-risk groups, such as those with congenital heart disease, connective tissue disease, portal hypertension, pulmonary disease, chronic pulmonary embolism, HIV infection and other underlying diseases, those taking diet pills, central appetite suppressants, and those with a family history of idiopathic or hereditary disease. 2.Echocardiography. 3.Perform right heart catheterization. Treatment 1, β-blockers make myocardial contraction weaken, reduce outflow tract obstruction, reduce myocardial oxygen consumption, increase diastolic ventricular dilation, and can slow down the heart rate and increase the heart beat volume. Propranolol was first used, starting with 10 mg each time, 3~4 times/d, and gradually increasing the dose to improve the symptoms but not low heart rate and blood pressure, up to about 200 mg/d. But recently it was found that β-blocker therapy cannot reduce arrhythmias and sudden death, and does not change the prognosis. 2, calcium antagonists both negative inotropic effect to weaken myocardial contraction, and improve myocardial compliance and favor diastolic function. Verapamil 120~480mg/d, divided into 3~4 times orally, can make long-term relief of symptoms, and be used with caution for those with low blood pressure, sinus function or atrioventricular conduction disorders. Diltiazem treatment is also effective, with a dosage of 30-60mg, 3 times/d. The combination of beta-blockers and calcium antagonists can reduce side effects and improve the efficacy.