Thickening or calcification at the tendon or tendon stop is a clinical condition due to tendinopathy disease. Tendinopathy was first proposed by German scholars in the 1940’s. In 1986, Perugia noted in his work that “there is a great contradiction in using the term inflammation (itis) as a suffix to describe degenerative changes in tendon tissue”. Therefore, the primary goal of the physician when employing treatment should be to block collagen breakdown, not to reduce inflammation. This requires a considerable period of rest and treatment to interrupt the cyclic cycle of tendinopathy, promote collagen production and maturation, and restore tension and functional strength to the tendon. How to effectively prevent thickening or calcification at the tendon or tendon stop? (a) Treatment 1. Educate patients: Doctors must patiently explain to patients and coaches the pathological changes and possible consequences of tendinopathy. Collagenous lesions are very difficult to treat, and in addition to pain and affecting movement, the brittleness of the tendon increases and the possibility of rupture increases several times. Therefore, a period of rest is necessary to warn those athletes who continue to participate in the sport after warming up. 2. Biomechanical load shedding: As the disease is often associated with local overload of the tendon and training errors. Therefore, it is very important to check the equipment used such as running shoes and rackets, check the movement mechanics such as running, throwing and power posture, diagnose and correct the possible muscle imbalance, all of the above. 3, anti-inflammatory measures: commonly used cold therapy, electrotherapy, cortisone injection, etc. Cold therapy is necessary for tendinopathy because there are many new blood vessels in tendinopathy tissue, and cold therapy can constrict the blood vessels. Electrotherapy mainly uses laser, high voltage electrical stimulation, etc. Electrical stimulation under laboratory conditions can stimulate collagen synthesis, which is effective for tendinopathy treatment. There is no indication: NASID and cortisone are of any help in tendinopathy, but whether COX-2 inhibitors are effective is still under investigation. Cortisone injection therapy provides only short-term results, with symptoms returning quickly. It also inhibits collagen synthesis, leading to partial tears or complete rupture of tendon tissue. 4. Reduce the motor load: The use of braces or braces can reduce the mechanical load on the collagen fibers of the tendon and help in the treatment. Effective braces such as knee and elbow braces, heel pads, Achilles tendon and patellar tendon braces have been proven to be effective, and there are many that deserve further research. 5. Physical therapy and sports therapy: It is important to communicate with the physical therapist and rehabilitator to inform them of the pathological changes in the disease and to agree on the possible recovery time. According to the literature, the average treatment time may take 2 to 3 months to return to play during the first episode; for patients with chronic symptoms, it may take 4 to 6 months to achieve a therapeutic effect. The actual time depends on the person. 6.Appropriate strength exercise: It has been confirmed that centrifugal strength exercise is effective, probably because the special centrifugal gymnastics can stimulate the mechanoreceptors of tendon cells, produce collagen, help reverse the cycle of tendinopathy, and achieve therapeutic purposes. Animal experiments have confirmed that proper tendon loading helps collagen alignment and stimulates collagen cross-connection formation, both of which promote collagen tensile strength. 7.Surgery: As the last option for tendinopathy treatment, lesion tissue excision can be used with a success rate of 75% to 85% or better. A recovery time of 4 to 6 months may be required after surgery. (B) Prognosis The results are generally better when strict, scientific treatment and proper rest can be carried out. The surgical results are also better.