Herpes zoster, clinically known as herpes zoster, is an acute infectious skin disease caused by varicella-zoster virus infection, most commonly seen in the elderly or in people with suppressed immune function. The main manifestation is the appearance of clusters of herpes zoster in the corresponding skin segments of the unilateral sensory nerves of the body, often accompanied by localized neuralgia. The primary infection of herpes zoster is chickenpox, and the varicella-zoster virus is latent in the sensory ganglia after infection. If the body is subjected to certain stimuli, such as cold, excessive fatigue, trauma, x-ray exposure, HIV infection, and immunosuppressant applications, the immune function of the body is reduced. In this case, the latent virus is activated and travels down the sensory nerve axons, resulting in a band-like distribution of herpes, known as herpes zoster. Herpes zoster, which is difficult to determine if it is in the latent phase, can develop at any age and is often preceded by local skin sensory abnormalities, such as sensory hypersensitivity, burning pain, pins and needles, and neuralgia, days before the skin injury occurs. The most important clinical feature is a clustered rash arranged in bands along the peripheral nerves, mostly confined to one side of the body, with corresponding neuralgia at the site of the rash. Patients with herpes zoster should pay attention to early bed rest, avoid rubbing the lesion to rupture the rash, and prevent secondary infection. Under the guidance of a medical professional, topical treatment can be done with topical acyclovir cream and penciclovir cream. In case of secondary eye damage, you can use acyclovir eye drops, etc. If postherpetic neuralgia occurs, analgesics can be used.