Why gastroscopy is needed for cirrhosis

  Old Chen has been suffering from chronic hepatitis for some years, recently always feel some abdominal discomfort, bloating, abdominal pain vague attacks, along with appetite is not good, within a few days people lost weight.  Old Chen accompanied by his family came to the hospital, the doctor suspected that Lao Wu’s chronic hepatitis developed into liver cirrhosis. The doctor recommended that Lao Chen undergo a gastroscopy, but Lao Chen thought, “Isn’t that gastroscopy used to check gastrointestinal diseases, how can it be used to diagnose cirrhosis? Because after cirrhosis, the flow of veins from the esophagus and gastric fundus back to the liver is blocked and slowed, and as a result, the veins will swell and varicose, and when the venous pressure reaches a certain level, it will cause bleeding.  Upper gastrointestinal bleeding is the most common complication of cirrhosis and the most likely culprit of sudden death, as well as the main cause of death in cirrhosis.  In fact, the pathological changes are the same regardless of the cause of cirrhosis. Cirrhotic nodule formation and portal hypertension, with varices of the fundic esophagus developed from portal hypertension, is one of the most common complications (incidence of 40%-90%). The varicose esophagogastric veins are very easy to rupture due to various triggers (eating, overworking, weight-bearing or even forceful defecation), leading to upper gastrointestinal bleeding and life-threatening, which is the main reason for vomiting blood in cirrhotic patients.  In addition, whenever there are esophageal varices in the fundus of the stomach, there is definitely portal hypertension and cirrhosis, and doctors will also confirm the diagnosis of suspected cirrhotic patients through gastroscopy. Therefore, gastroscopy is of great significance in the diagnosis and treatment of patients with cirrhosis and chronic liver disease. Also gastroscopy is the easiest, effective, safe and economical means of treatment for upper gastrointestinal bleeding in some cirrhosis, such as gastroscopic esophageal variceal ligation, sclerosis, and tissue adhesive hemostasis.  Do gastroscopy to anticipate the possibility of bleeding It is understood that ruptured esophagogastric variceal bleeding is often aggressive, with a high mortality section for initial bleeding and a higher risk of rebleeding within 2 years in surviving patients. Therefore, it is extremely crucial to control the esophagogastric fundic varices and prevent and control their ruptured bleeding.  So, how should patients cooperate with prevention?  Patients with cirrhosis should have a gastroscopy to evaluate the esophagogastric fundic varices and the risk of bleeding through endoscopy before deciding whether to make further treatment. If the esophagogastric fundic varices are significant, or if there is already bleeding, the doctor will perform minimally invasive treatment (including ligation of the veins, injection of sclerosing agents, or tissue gel injection) under painless endoscopy to achieve the disappearance of the varices in the fundus and esophagus.  Cases of minimally invasive endoscopic treatment have been followed up, and patients have a very low probability of rebleeding 3-5 years after the procedure, so that not only the quality of survival of patients has improved, but also the incidence of death due to acute bleeding has been greatly reduced. Of course, patients need to be followed up regularly. For patients with hepatitis B or C, antiviral therapy, liver protection and enzyme-lowering therapy, as well as regular review of gastroscopy, which should be done at least once a year.  Can early stage cirrhosis be cured?  Many people think that cirrhosis is incurable and there is no room for maneuvering, but this is not entirely true.  Early cirrhosis still has a chance to be reversed, and the prognosis is different for different causes of cirrhosis. For example, cirrhosis caused by alcoholic liver, fatty liver, etc., after strict abstinence from alcohol and weight loss, the cause of the disease is removed, cirrhosis may be reversed. Therefore, it is necessary to stop drinking, and if you do not stop drinking, it is impossible to reverse the development trend of cirrhosis.  As for cirrhosis after hepatitis B and C, although antiviral treatment cannot reverse the course of cirrhosis, it can delay its development very well. On the contrary, if the replication of hepatitis virus is not controlled, the course of cirrhosis will be faster. The earlier the cause of the disease is intervened, the greater the chance of reversing early cirrhosis or delaying its progression.  In addition, patients should not rush to the doctor or abuse some drugs or supplements that can supposedly cure cirrhosis. Now there are some drugs for liver fibrosis, but the effect is not very exact; Western countries have been studying cirrhosis for 100 years, but no drug has been studied to find out what the effect is exact. The indiscriminate use of drugs will increase the burden on the liver, but may accelerate the progress of the disease.  In addition, patients with cirrhosis should regularly review liver ultrasound or CT examinations in order to achieve early detection and early treatment of cirrhotic carcinoma, so that the survival rate of patients with cirrhosis can be fundamentally improved.