This type is characterized by difficulty in suppressing involuntary movements of the limbs with the will, and it affects the whole body, including the facial muscles, and is often accompanied by language impairment because the vocal and phonological organs are also involved. Most of the patients with a history of neonatal asphyxia and nuclear jaundice are suffering from tardive dyskinesia. Because the upper extremities are more damaged than the lower extremities, many children use their feet to write. The lesion is in the deep basal nucleus of the brain, and the external cone pathway is predominant, accounting for about 20% of cerebral palsy. The main symptoms of abnormal development in children with hand-footed cerebral palsy: a. Backward motor development and reduced active movement Among the symptoms of pediatric cerebral palsy, the backward motor development is manifested in two aspects: gross motor and/or fine motor. There are many indicators to determine whether the motor development is backward, and each movement is different in different years (months), but some main indicators should be mastered in clinical application. Normal children can lift their heads at 3 months of age, reach out and touch things at 4-5 months of age, hold their hands in front of their chest, and play with their hands in front of their eyes when they are quiet. At 6-7 months of age, the child can sit alone on a hard bed surface without falling, and at 8-10 months of age, the child can crawl and move both upper and lower limbs alternately. Children with cerebral palsy generally do not reach the level of normal children at these ages. During the neonatal period, children with cerebral palsy often show reduced movement, poor sucking ability and poor feeding response. Normal 3-month-olds often have kicking and stomping-like movements in the supine position, and alternate kicking and stomping. In normal 4-5 month olds, the upper limb activity is very flexible, but in children with cerebral palsy, the upper limb activity is also reduced. The normal child has not yet formed right or left lee within the age of 1 year, while the hemiplegic type of spastic cerebral palsy shows that he often uses only one hand to hold or touch things, and the activity of the other hand is reduced, and the hand is often in the shape of a fist. Muscle tone is the tension of muscles in a quiet state. The muscle tone can be understood by passively flexing, straightening, rotating the front and back of the limb. Small infants can hold their forearms and shake their hands to understand the muscle tone of the upper limbs according to the range of motion of the hands. To measure the muscle tone of the lower limbs, you can also hold the lower leg and shake its foot, and judge its tension according to the range of foot activity. The muscle tone can also be checked by the “pull test”, which is easy to grasp, holding the child’s hands, pulling them from a supine position to a sitting position, and observing the back hanging of the head to understand the muscle tone of the back of the neck. When examining muscle tone, it is important to note that some older children with a longer course of disease may have joint contractures that restrict joint movement, which should not be mistaken for increased muscle tone. The abnormal posture of children with cerebral palsy is diverse and is related to abnormal muscle tone and delayed loss of primitive reflexes. In the first year, they often lie quietly and have almost no voluntary movement. In the supine position, their posture is the opposite of the spastic type, showing lower limb flexion, hip abduction and ankle dorsiflexion. When picked up in the upright position, the head can be controlled in the central position. The deep reflexes (knee reflex, biceps reflex, Achilles reflex, etc.) of children with spastic cerebral palsy are active or hyperactive, and sometimes ankle clonus and Babinski’s sign can be induced. The neurological reflexes of children with cerebral palsy often show delayed disappearance of primitive reflexes and weakened or delayed appearance of protective reflexes.