Elevated uric acid can cause a series of health hazards. When the blood uric acid is elevated > 480 μmol/L, the uric acid in the blood is already in a saturated state, and urate crystals are easily formed at this time. Uric acid deposits in the joints can lead to gouty arthritis, and gouty arthritis, if repeated, can seriously affect the quality of life of patients. When hyperuricemia occurs, urate can be deposited directly in the kidneys, causing acute or chronic uric acid inflammatory nephropathy and uric acid kidney stones, which can lead to end-stage renal disease, i.e. uremia, in severe cases. In addition, hyperuricemia is also closely related to the occurrence of hypertension, diabetes, coronary heart disease, and stroke. Hyperuricemia can lead to hypertension. Long-term hyperuricemia leads to vascular endothelial dysfunction, activates the renin-angiotensin system, induces proliferation of vascular smooth muscle cells and leads to hypertension. Elevated serum uric acid levels also have an impact on insulin resistance and the development of diabetes. Uric acid affects insulin metabolic signaling by affecting the renal tubules and decreases the body’s sensitivity to insulin. Studies have found that for every 60 μmol/L increase in uric acid levels above the normal range, the risk of developing diabetes doubles. Hyperuricemia is also closely related to the development of coronary heart disease. Hyperuricemia can directly damage the vascular endothelium, while making platelets dysfunctional and prone to adhesion and aggregation, leading to coronary heart disease. For every 60 μmol/L increase in uric acid above the normal range, the risk of coronary heart disease increases by 1.5 times. Therefore, elevated uric acid is very detrimental to health and needs to be avoided.