Having trace ascites is not necessarily cirrhosis. There are many causes of micro ascites, such as tuberculous peritonitis, cirrhosis, nephrotic syndrome and so on. 1. Tuberculous peritonitis: Mycobacterium tuberculosis invades blood vessels and intestinal wall, causing impaired absorption of body fluids and inflammatory exudation, resulting in exudative ascites. In addition to ascites, there are manifestations such as abdominal distension, diarrhea and tenderness of abdomen. 2. Cirrhosis: (1) Portal hypertension: lymphatic fluid production increases in cirrhosis, and when it exceeds the drainage capacity of the catheter, the lymphatic fluid leaks from the hepatic peritoneal preparations into the abdominal cavity; the hydrostatic pressure of the portal vein increases, which causes the fluid to enter the tissue interstitial space, thus forming ascites. (2) Plasma colloid osmotic pressure: when liver function decreases in cirrhosis, the ability to synthesize albumin decreases, and hypoproteinemia occurs, which leads to the entry of intravascular fluid into the tissue interstitium, causing ascites. (3) Insufficient effective circulating blood volume: liver blood output increases in cirrhosis, which dilates the visceral arteries, and a large amount of blood is retained in the blood vessels, resulting in insufficient effective circulating blood volume, increased aldosterone secretion, and sodium and water retention, which produces ascites. 3. Nephrotic syndrome: when renal function declines, the ability of the kidneys to drain water and sodium decreases, resulting in water and sodium retention; prolonged proteinuria leads to hypoproteinemia, and plasma colloid osmotic pressure decreases, causing ascites. There are many other causes of microscopic ascites, such as chronic hepatitis, urological tumors, etc. It is recommended to consult a doctor in time for a clear diagnosis and targeted treatment.