Lack of rehabilitation on muscle It is well known that braking can lead to costal muscle atrophy. Studies have found that Class I fibers, which have a high metabolic demand in the early stages of braking, are the first to atrophy and are significantly more severe than Class II fibers, but as the braking time increases, both Class I and Class II fibers atrophy, and there is no significant difference in the degree of atrophy between the two. Some scholars also found that muscle atrophy has a great relationship with the location of limb fixation. If the muscle is fixed in a shortened position, the number of muscle segments will be reduced by up to 40%, while the length of the muscle segments will also be reduced, and the reduction of the number and length of the muscle segments will be significantly delayed. Therefore, how to ensure the braking necessary for the repair of sports trauma, at the same time, the most maximum early activity, early rehabilitation treatment and prevention of muscle atrophy are the basic topics of trauma rehabilitation. The effect of lack of rehabilitation treatment on articular cartilage and bone The process of alternate compression and decompression of articular cartilage is stopped after joint braking, which leads to the obstruction of nutrient metabolism of articular cartilage. The imbalance in muscle tone, the continuous compression of certain parts of the articular cartilage, the excessive loss of fluid components of the cartilage matrix there, and the failure to exchange nutrients well can lead to cartilage degeneration and necrosis and exfoliation. Joint fixation can also lead to the proliferation of fibrofatty connective tissue that adheres to the surface of the articular cartilage, blocking the dissipation channels for nutrients. Longer periods of non-weight bearing or poor weight bearing can lead to bone atrophy, osteoporosis, and fragility fractures. Lack of rehabilitation on ligament and joint mobility Ligament injury or surgical repair often requires splinting or plaster fixation, and studies have found that collagen fiber density decreases after ligament fixation; the diameter of the fiber bundle shrinks; the cross-section of the ligament shrinks, the strength of the ligament decreases, and decalcification and bone resorption occur at the ligament stop. Furthermore, the recovery of ligament properties after unfixation takes a considerable amount of time, with studies showing that it often takes several months to restore compliance after 8 weeks of fixation and 12 months to restore its strength and stiffness. Reduced ligament movement after fixation can lead to ligament adhesions and, if fixed in the ligament and joint laxity position, can also lead to contracture of the ligaments and joint capsule, resulting in decreased joint mobility. Joint mobility is closely related to the flexibility and suppleness of the joints, muscles, ligaments and joint capsule. Joint mobility disorders are divided into two categories: bony and fibrous. Bone mobility disorders are difficult to treat because of joint deformity or joint fusion caused by injury or disease; fibrous mobility disorders are caused by contracture or adhesion of soft tissues inside and outside the joint. After sports trauma, due to the injury of joints, muscles, ligaments and joint capsule, pain and muscle spasm; and the shortening and contracture of muscles, ligaments and joint capsule after braking, scarring and adhesions of peri-articular tissues often lead to the limitation of joint mobility, mostly belong to fibrous mobility disorders, which can be effectively treated by joint mobility exercises and manipulation and release.