Patients with cerebral infarction who faint are in a more serious condition and are mostly considered to be caused by lesions in the vertebrobasilar system. Patients may also have difficulty swallowing and choking on water, and may also develop nystagmus and diplopia, and may even cause limb palsy and bulbar palsy. In severe cases, there may also be impaired consciousness, including coma, decortication and vegetative state, and unstable vital signs may also occur, which should be treated promptly and comprehensively. Consider intravenous thrombolytic therapy if the patient is suitable for thrombolysis in the ultra-early stage, and antiplatelet coagulation, neuroprotection, and improvement of blood circulation when thrombolysis is not suitable. If the patient has swallowing dysfunction, it is necessary to lower the gastric tube and retain the gastric tube for nasal feeding diet and nasal drug administration when it is difficult to recover in the short term. Most patients may have some sequelae, and some patients may be clinically cured with active and correct treatment.