Secondary hyperuricemia is potentially curable, whereas primary hyperuricemia is generally not.
Hyperuricemia is divided into primary hyperuricemia and secondary hyperuricemia. Primary hyperuricemia generally refers to hyperuricemia caused by genetic or congenital enzyme defects, excessive alcohol intake, enhanced nucleic acid metabolism, and high purine levels, etc. This type of hyperuricemia is generally incurable and needs to be controlled by diet or medication (e.g., phenylbromarone, etc.).
Secondary hyperuricemia refers to the elevation of blood uric acid caused by blood system diseases, kidney diseases, drugs and excessive production of organic acids, etc. This type of hyperuricemia may be cured after the patient is actively treated for the primary disease or the triggers are removed.
Patients with hyperuricemia can go to endocrinology, rheumatology and immunology departments of regular hospitals for detailed consultation.