Late-onset dyskinesia has a low probability of self-healing after discontinuation of medication, and prevention is the main clinical goal. Late-onset dyskinesia is an abnormal involuntary movement associated with long-term use of dopamine receptor blockers, which can involve the face and neck, causing tongue thrusting, chewing, pouting, crooked jaws, or turning of the neck, and the limbs and trunk, which can be characterized by choreiform movements. The most common etiology is antipsychotic drug use. The specific mechanism of tardive dyskinesia is unknown and there is a lack of effective medications for the treatment of tardive dyskinesia, resulting in a low probability of clinical cure, with prevention as the primary goal. Common first-line therapeutic agents include deuterated buprenazine and pentylphenazine, and other potentially effective oral medications include clonazepam, amantadine, and ginkgo biloba extract. Local injections of botulinum toxin and deep brain stimulation methods may be effective for patients who do not respond well to oral medications. Delayed movement disorders have a low probability of resolving spontaneously after discontinuing medication, and should be treated and intervened under the guidance of a medical professional to improve quality of life. The following is an example of a patient with delayed dyskinesia.