Myocardial abscess is one of the main clinical symptoms of dilated cardiomyopathy (cardiomyopathy (DDM)), a group of lesions that cause progressive dysfunction of the heart due to structural changes in the lower portion of the heart’s chambers (i.e., ventricles) and impaired function of the myocardial wall. Its clinical manifestations include heart enlargement, arrhythmias, embolism and heart failure. The causes are generally related to viral infections, autoimmune reactions, genetics, drug toxicity and metabolic abnormalities. (According to the pathology, it can be divided into dilated cardiomyopathy, hypertrophic cardiomyopathy and restrictive cardiomyopathy, etc.). Differential diagnosis of myocardial abscess: Myocardial infarction refers to the interruption of blood flow in the coronary arteries on the basis of coronary artery disease, resulting in severe and prolonged acute ischemia of the corresponding myocardium, which ultimately leads to ischemic necrosis of the myocardium. Myocardial ischemia Myocardial ischemia is caused by insufficient blood supply to the heart. The main manifestations are: palpitations, discomfort in the heart region, sometimes heart throbbing pain or discharge colic; shortness of breath, exercise, fullness or agitation is more serious, weakness; in severe cases, it can be short-term shock. Gray and flaccid myocardium A type of myocardial damage that may be associated with immune myocardial damage that occurs after viral infection and is usually seen on ultrasound in dilated cardiomyopathy. It may be used as a differential diagnosis from other cardiomyopathies. Myocardial stunning, also known as post-ischemic myocardial dysfunction, is a phenomenon in which transient myocardial ischemia has not yet resulted in myocardial necrosis, but mechanical dysfunction after reperfusion to restore normal blood flow takes hours, days, or weeks to fully recover. Myocardial hypertrophy This is a slower but more effective compensatory function that occurs mainly in cases of chronic stress overload, where the total amount of myocardium increases and contractility is strengthened, allowing the heart to maintain normal blood circulation while having a considerable reserve capacity. However, this compensatory function also has its disadvantages, mainly because the hypertrophied myocardium requires increased oxygen, and the coronary artery blood supply is often unable to meet, resulting in myocardial ischemia, which will eventually lead to myocardial contractility loss. Hypertrophic cardiomyopathy is characterized by myocardial hypertrophy. It is characterized by hypertrophy of the ventricular myocardium, typically in the left ventricle, more so in the interventricular septum, and occasionally by concentric hypertrophy.