Pseudotuberculous nodules are lesions caused by worm eggs in schistosomiasis. Acute worm egg nodules are an acute necrotizing, exudative lesion caused by mature worm eggs. After 10 days of acute worm egg nodules, the trichomes in the eggs die, the eggs and necrotic material are removed, absorbed or calcified, and the macrophages in the lesion diffuse into epithelioid cells and foreign multinucleated giant cells, forming granulomas similar to those of tuberculous nodules, which are known as pseudotuberculous nodules, i.e., chronic worm egg nodules. As a result of repeated infections, the female worms kept laying eggs, which were deposited in batches in the intestinal wall, and the lesions varied in age and newness. On the basis of fiber thickening, chronic ulceration and polyp formation, there is a possibility of cancer. The examination methods are as follows. Liver lesions caused by schistosomiasis are early hepatomegaly with corn-like yellow granules (worm egg nodules) visible on the surface. In the late stage, due to the proliferation of a large number of fibrous tissues around the portal vein branches, the liver hardens and shrinks, and there are nodules of varying sizes on the surface, forming schistosomal cirrhosis, and the establishment of portal collateral circulation makes the veins of the lower end of the esophagus and the fundic veins of the stomach varicose, and the spleen is enlarged due to the siltation and swelling of portal hypertension, which can lead to hyper-splenic function. The ectopic damage caused by schistosomes is most common in the lungs and brain, with cornified nodules and perinodular exudative inflammation seen in the lungs, while the brain may show nodules of eggs and glial cell hyperplasia at different periods.