Lumbar disc herniation is one of the common diseases in orthopedics, and about 1/5 of patients with low back pain are caused by lumbar disc herniation. It has been more than seventy years since Mixterher and Barr proposed this disease in 1934. From the epidemiologic analysis at home and abroad, the population rate and absolute value of its incidence are on the rise. The age of onset ranges from a few years to tens of years, and we have seen patients with lumbar disc prolapse as young as 9 years old. The rise in the incidence of this disease is related to changes in the environment we live in, our living and working habits. Chronic poor back habits are the main cause. In the past seventy years, the majority of medical workers have been working hard on research and have made great progress both in the familiarity of the disease and treatment methods. From folk remedies, Chinese herbs, to traction massage physiotherapy, reset and so on intervention, minimally invasive, surgical treatment, treatment methods are endless and varied. How can we choose among the many treatment methods for individual patients? Patients are clueless, but also many doctors do not know, full of confusion. In fact, the treatment of the disease, the choice of indications is the key. That is to say, in the vast sea of treatment methods, understand the degree of their own condition, choose the most targeted treatment methods, lumbar disc herniation can be completely cured very quickly. Here to years of clinical specialty experience to open post, guide your patients for symptomatic treatment, in order to reduce the patient’s confusion, no longer treatment on the detour. Due to my shallow knowledge, extraordinary and authoritative experts at home and abroad compared to the gap is still very large, to consult the appropriate comments just to provide reference, not for the final conclusion. The pathology of lumbar intervertebral disc herniation The intervertebral disc organization itself lacks blood supply, and the repair ability is extremely poor, coupled with the negative heavy activities. Generally after the age of 20, the intervertebral disc begins to undergo degenerative changes, and the toughness and elasticity of the annulus fibrosus gradually decreases. At this time, if the trauma `especially cumulative strain injury, it becomes the cause of the rupture of the annulus fibrosus. In many cases, there is no history of trauma, but after catching a cold, the tension in the muscles and ligaments increases, which increases the internal pressure of the disc and promotes the rupture of the atrophied annulus fibrosus. The intervertebral disc is a remarkable structure made of connective tissue that is burdened with unique functions. Any alteration of the intervertebral disc affects its normal mechanical efficiency or interferes with its normal function of balancing, absorbing and redistributing its forces to the spine. The intervertebral disc consists of the nucleus pulposus, the annulus fibrosus, and the cartilaginous plate. The nucleus pulposus of the intervertebral disc contains a small amount of collagen fibers in addition to a soft matrix of mainly mucopolysaccharides. The nucleus pulposus accounts for more than half of the volume of the intervertebral disc, and because it is characterized by deformability, it is able to transmit load forces appropriately. The ability of the intervertebral disc to maintain proper function is closely related to its water content water volume, which in turn is stabilized by the content of polysaccharides. The annulus fibrosus is differentiated from the nucleus pulposus, although still significant. The collagen fibers of the annulus fibrosus are in the form of dense laminae, with the fibers of each layer interlacing with each other at right angles to each other and with the vertebrae at an angle of 45°, and this laminar structure accommodates the stresses of pressure and tension as well as the flexion and rotational stresses caused by the spinal column. The cartilage plate is glassy cartilage, which is scarfed between the vascularized vertebral cavernous bone and the avascular nucleus pulposus. On the surface of the glassy cartilage, the collagen fibers are parallel to each other on the surface and perpendicular to each other in the deeper layers near the bone. Proteoglycans ground an important part of the intervertebral disc matrix and are important structures for the mechanical and chemical function of the intervertebral disc. Proteoglycans are large molecules, extremely viscous, and very hydrophilic. Under normal conditions, the nucleus pulposus is highly compressible and has a strong loading capacity due to the properties of proteoglycans. If the glycan chains of the proteoglycans break down, they lose their ability to retain extracellular water. The biochemical integrity of the nucleus pulposus amidships is determined by its water-containing volume. Normally the intervertebral discs are under pressure and redistribute their forces to the spine and are an important part of accomplishing normal function. The formation of a herniated disc is normal with an excess of proteoglycans, which will cause the nucleus pulposus to fluid and increase, and the pressure within the nucleus pulposus to rise, predisposing the disc to herniation. However, mucopolysaccharides in the nucleus pulposus can produce a new equilibrium through reduction and reintegration. Protein polysaccharide progressive reduction, can promote collagen fibrosis, the nucleus pulposus due to collagen deposition, fibrosis increased and gradually lose the original compressibility and load capacity, the nucleus pulposus of the intervertebral disc at any time need to be in the weight-bearing situation to absorb and will be the stress redistributed to the spinal column to go to the function will not be able to do, thus causing disc injury. If external trauma or excessive stress is added to the damaged disc, it is more likely to cause disc herniation. It is believed that glycoproteins and beta-proteins in the matrix of the nucleus pulposus form the antigen of autoimmunity, and it is the release of this antigen (referring to the release of beta-proteins from degenerative discs and herniated discs, which are normally encapsulated in the nucleus pulposus) that causes persistent stimulation of the organism, resulting in an immune response, and at the same time causes inflammatory reactions in the nerves, which results in pain.