1.What is coronary heart disease? Coronary heart disease is the full name of “coronary atherosclerotic heart disease”, the basic lesion is the coronary artery wall of the atherosclerotic plaque blocking part or all of the coronary artery lumen, resulting in coronary artery stenosis, resulting in the obstruction of blood flow, the occurrence of myocardial ischemia. The consequences are: (1) ischemia of cardiomyocytes, resulting in a weakening of their contractility, when the patient has angina pectoris attack. (2) Myocardial cell necrosis, resulting in myocardial cell loss of contractile function, clinically known as myocardial infarction (“infarction”). Necrosis of cardiomyocytes can lead to papillary muscle rupture, septal perforation, or rupture of the ventricular wall. (3) Myocardial cells are neither necrotic nor contractile, but can form “ventricular wall tumor”, which seriously affects the heart function. 2.What are the symptoms of coronary heart disease? There are many symptoms of coronary heart disease, the common ones are: 1. Clinical data There were 5 cases in this group, aged 36-70 years old (average 53.8 years old), among which 4 cases were male and 1 case was female. 5 patients were diagnosed with chronic obstructive pulmonary emphysema according to the medical history and chest thin-layer CT scan before the operation, and there were 4 cases of herpetic emphysema, and 1 case of homogeneous pulmonary emphysema. One female patient had consolidated pneumoconiosis with right-sided pneumothorax, one male patient had consolidated bilateral silicosis with left-sided fluid pneumothorax, one male patient had consolidated right-sided pneumothorax with left-sided destroyed lung, and one had consolidated left-sided pneumothorax. According to the revised dyspnea index grading by the American International Medical Research Council, 4 cases were grade 5 and 1 case was grade 4. All patients were treated conservatively in respiratory medicine for more than 3 months-5 years before surgery, and those with combined pneumothorax underwent closed thoracic drainage. There were 4 cases who required continuous oxygen intake before surgery, and 1 case of intermittent on-demand oxygen intake. All patients were unable to take care of themselves. The preoperative results of patients’ pulmonary function, peripheral oxygen saturation (noninvasive) and blood gas analysis (without oxygenation) are shown in the attached table. 2 Results There were no surgical deaths in this group. The median postoperative time to air leakage was 2.5 d, and the median time to intubation was 5 d. There were no cases of postoperative respiratory failure. Two cases of postoperative lung infection on the affected side and one case of acute myocardial infarction were cured after active treatment. The follow-up rate was 100% for 1-8 months after the operation, and there were no recurrence of pneumothorax and death cases. All patients’ dyspnea symptoms improved significantly, graded by dyspnea index: 3 cases were grade 3, 2 cases were grade 2. Postoperative pulmonary function and arterial blood gas analysis were significantly improved compared with the preoperative period.