[Overview] Mycosis fungoides (monilialormycoticvaginitis) is caused by mycobacterial infection. Its incidence has been higher than that of trichomoniasis. The medical term for mycotic infection is Candida infection, so mycotic vaginitis is also known as Candida vaginitis is most commonly seen in young girls, pregnant women, diabetic patients, and patients who have been treated with larger doses of estrogen after menopause. The most common symptoms of Candida infection are leucorrhea, burning and itching of the vulva and vagina, exogenous urinary difficulties, and vulvar map-like erythema (mycosis or Candida vulvovaginitis). The typical leukorrhea is curd-like or flaky, the vaginal mucosa is highly erythematous and white thrush-like patches are seen attached and easily peeled off, underneath which is the erosive base of the damaged mucosa, or shallow ulcers are formed and in severe cases petechiae may remain. The leucorrhoea does not always have these typical features, but can range from aqueous to curd-like leucorrhoea, and in some cases is entirely a thin, clear, steep plasma exudate that often contains white flakes. The itching symptoms of mycosis vaginalis during pregnancy are particularly severe, even sitting and lying down, painful and unusual, and there can also be symptoms such as frequent urination, painful urination and painful intercourse. In addition, there are about 10% of women and 30% of pregnant women who are carriers of mycobacteria but have no clinical manifestations. Change the pH of the vagina, such as alkaline drug douche vagina, 2-4% soda douche vagina, to change the living environment of mycobacteria. (1) Mycobacterium (nestatin) vaginal pessary (containing mycobacterium 250,000 U), inserted deep into the vagina, once in the morning and once in the evening or once every night for 2 weeks. (2) Oral administration of mycoplasma 500,000 U 4 times daily. (3) Topical application of compounded mycoplasma cold cream, 2 times a day. (4) Oral ketoconazole (ketoconazole) 400mg twice a day for 5 days. (5) Trichomycin (trichomycin), see treatment of trichomoniasis for details. In addition, oral clotrimazole (clotrimazole) or chlorambucil 0.5-1g 3 times a day, or topical 1-5% ointment cream or application, 3-4 times a day, is also effective. (6) 1 to 2% gentian violet solution topical application, is a long-established therapy, also very effective, widely used, but has the disadvantage of contamination of underwear. If a pregnant woman has mycosis vaginalis, it may heal itself after delivery, but the newborn is at risk of infection, so timely treatment is still needed. 3. For male carriers must also be routinely treated, which is one of the important measures to prevent female patients from relapsing. Etiology】 There are many kinds of mold (Fungus), the most important one in human body is Candida albicans. Vaginal infections are caused by Candida albicans in 80-90% of cases, the rest are other species of Candida and ball-like yeast (Tomlopsis), so mycosis vaginitis is actually Candida vaginitis or vaginal candidiasis (vagmalcandidiansis). Candida albicans is ovoid in shape and consists of budding spores and cells that germinate and elongate to form pseudomycorrhizae, which are connected to the spores in a branched or chained form. Candida is usually a spoilage parasite that can live in the skin, mucous membranes, digestive tract or other organs of the normal human body, and is often present in the vagina without symptoms. About 10% of non-pregnant women with increased leucorrhea and about 30% of pregnant women have this bacteria parasitic in the vagina. When vaginal glycogen increases and acidity rises, or when the body’s resistance decreases, it can be the cause of the disease. Long-term application of broad-spectrum antibiotics and adrenocorticosteroids can greatly increase mycobacterial infections. Because these two drugs can lead to dysbiosis in the organism, changing the interrelationship between microorganisms in the vagina and decreasing the ability to fight infection. In addition, vitamin deficiency (vitamin B complex), severe infectious diseases, and other wasting diseases can become favorable conditions for Candida albicans to multiply. During pregnancy, the glycogen content of vaginal epithelial cells increases, the acidity of the vagina is enhanced, and the renal sugar threshold of pregnant women decreases, often with nutritional diabetes, and the sugar content in the urine increases and promotes the growth and reproduction of Candida albicans.