Epilepsy is somewhat hereditary. It is also known as idiopathic epilepsy, an epilepsy of unknown etiology, which is currently clinically favored as being caused by genetic mutations and certain congenital factors with a clear genetic predisposition. A key feature of idiopathic epilepsy is that, to date, humans have not identified structural damage or biochemical abnormalities in the brain that would be sufficient to cause seizures, and that molecular biology needs to be applied to discover the cause of the epilepsy. Epilepsies whose cause is still unclear are commonly referred to as idiopathic epilepsies. Familial partial epilepsy with different foci is an autosomal dominant inheritance. Genetic linkage analysis revealed that the mutated gene is located on chromosome 2. Familial temporal lobe epilepsy, a familial epilepsy recently endorsed by the International League Against Epilepsy, is probably an autosomal dominant inheritance with an episodic rate of 60%. Familial partial epilepsy with auditory symptoms. Its candidate gene is located on the short arm of chromosome 10. Benign familial neonatal convulsions, the first primary epilepsy to be successfully analyzed by chain analysis.