The hazards of poor coagulation function include: causing spontaneous bleeding manifestations, such as intracranial hemorrhage, joint hematoma, ecchymosis and petechiae of skin and mucous membranes, etc.; and difficulty in hemostasis of wounds caused by trauma and surgery.
Poor coagulation function is commonly understood to mean that the blood cannot form blood clots normally to stop bleeding, and the routine examination of coagulation can show prolongation of prothrombin time, partial thromboplastin time, and obvious elevation of INR.
Poor coagulation can be caused by a lack of or excessive consumption of coagulation factors in the blood, overdose of anticoagulant drugs such as warfarin, and many other etiologic factors.
In less severe cases, poor coagulation may only be manifested as a significant prolongation of the time needed to stop bleeding, such as trauma-induced wounds or surgical wounds with persistent active bleeding and difficulty in stopping bleeding.
In severe cases of poor coagulation, spontaneous bleeding may occur, which may be characterized by different bleeding manifestations, such as pin-prick hemorrhagic spots on the skin, bruises, etc., and spontaneous intracranial hemorrhage may occur in some patients. Due to congenital deficiency of coagulation factor VIII or IX, hemophiliacs often have hematoma of joints, bleeding gums, bleeding from the nose, blood in the stool and other manifestations.
Patients with poor coagulation function need to avoid strenuous activities and go to the hematology department in time to improve the relevant examinations to clarify the specific causes of the disease, and then under the guidance of the doctor for the cause of the treatment, and if necessary, can be transfused with coagulation factors to support the treatment.