The sudden growth of acne on the face is clinically known as common acne, and the occurrence of acne is closely related to overproduction of sebaceous glands, blockage of follicular ducts, bacterial infections, and inflammatory reactions. The majority of this type of disease occurs during adolescence, mainly because the level of androgens, especially testosterone, secreted by the body rises rapidly when entering adolescence. Elevated levels of testosterone promote the development of sebaceous glands and the production of large numbers of sebaceous glands. At the same time, the ducts at the hair follicles can easily become clogged, and once the sebaceous glands are not drained they can form keratin plugs, also known as microcomedones. Within the hair follicles are a variety of microorganisms, especially Acinetobacter. When P. acnes multiplies, the esterase secreted by P. acnes produces free fatty acids, which produce both chemokines and inflammatory mediators, leading to localized inflammation, which is clinically known as acne.