Cerebral palsy (cerebral palsy) is a comprehensive name for non-progressive central motor dysfunction caused by multiple causes. In severe cases, in addition to limb paralysis, there is also mental retardation, involuntary movements and functional impairment of hearing, vision and speech, etc. accompanied by convulsions (epilepsy). According to research statistics, 60-75% of them are spastic (hypertonic type). 1. Pre-birth factors: fetal infection, hemorrhage, hypoxia and developmental malformations and maternal gestational hypertension, diabetes, traumatic brain injury and radiation, etc. Genetic inheritance is only occasional; 2. Perinatal factors: amniotic fluid blockage, fatty fecal inhalation, umbilical cord wrapping around the neck and other causes of asphyxia, or injuries during delivery, intracranial hemorrhage, hypoxia, etc. 3.After birth: cerebral hypoxia, cholestatic encephalopathy (yellow bile too deep), intracranial hemorrhage, serious infection, etc. occur during the neonatal period. In recent years, cerebral palsy is more common in premature infants. It is related to vascular damage and concurrent metabolic disorders. Clinical classification 1. Spastic type: 75% of the cases, often mixed with other types. Bilateral: the lower limbs are heavier than the upper limbs. When laughing and standing, the two lower limbs are spasmodically straightened and the upper limbs are stretched backward. 2, movement disorder type: 20% of all patients, appear purposeless, involuntary movements, or hand and foot restlessness, chorea, excessive movements, (disappeared during sleep), muscle tremor. 3, ataxia type: accounting for 1-2%, mainly manifested as gait instability, low muscle tone. It is not easy to be noticed in the neonatal period and is often detected by intraocular strabismus. Most of them develop bilateral or limb movement disorders within a few months to a year after birth. CT examination 1, asphyxia caused by central atrophy or cortical atrophy; 2, bilirubin encephalopathy, less often seen CT changes or mild atrophy; 3, premature infants have central atrophy, lateral ventricular central part of the abnormal, peripheral segmentation atrophy, etc., birth injury is seen with low absorption signs such as cavitation; 4, the cause of unknown. Half of the CT cases showed normal performance, but cortical atrophy or ventricular enlargement was also seen; 5. Acquired causes of CT changes were hydrocephalus and cerebral atrophy. In conclusion, the abnormality rate of CT is proportional to the degree of paresis, the younger the age, the higher the abnormality rate of CT, and with age, the new abnormalities seen at the initial diagnosis gradually become smaller and disappear or normal at the age of 2-3 years. Treatment 1, physical exercise and physical therapy: including sunlight air, water therapy, muscle training and limb exercise, as an important part of the treatment, massage, acupuncture, massage and chiropractic contracture therapy and electrotherapy, phototherapy are helpful. 2, drugs: early use of brain activator; 3, orthopedic surgery: (for spasticity) (1) muscle tone adjustment (tendon lengthening, or tightening or transposition); (2) functional reconstruction (tendon transposition or nerve transposition); (3) high selective severance of nerve roots in the anterior horn of the spinal cord; (4) orthopedic bone and joint.