The global prevalence of alcoholic liver disease shows a continuous increasing trend. There is a lack of national large-scale epidemiological survey data on alcoholic liver disease in China, but regional epidemiological surveys show that the number of people who drink alcohol and the prevalence of alcoholic liver disease in China are on a rapid rise. Epidemiological surveys in northern China showed that from the early 1980s to the early 1990s, the proportion of alcoholics in the general population rose from 0.21% to 14.3%; at the beginning of this century, epidemiological surveys in southern and central and western provinces showed that the drinking population increased to 30.9%~43.4%. The proportion of alcoholic liver disease in hospitalized patients with liver disease in the same period has also been increasing, from 4.2% in 1991 to 21.3% in 1996; studies from the Thirty-second Hospital of the People’s Liberation Army reported that the proportion of patients hospitalized with alcoholic liver disease increased by 170% in more than ten years from 2002 to 2013. Thus, it is clear that alcohol-induced liver damage has become a problem that cannot be ignored in China. Alcohol abuse and dependence significantly increases the risk of developing alcoholic cirrhosis, most significantly in people aged 40 to 59 years. It has been previously reported that long-term daily alcohol consumption, especially white wine or beer, is more likely to cause alcoholic cirrhosis. The results of a retrospective study showed that the 5-year liver disease-related mortality rate was 13% in patients with early/compensated alcoholic liver disease and up to 43% in patients with decompensated disease; the long-term prognosis of patients with early/compensated alcoholic liver disease was influenced by the stage of liver fibrosis, with severe fibrosis (F3/4) having a significant impact on the 10-year mortality rate; for patients with decompensated alcoholic liver disease, clinical characteristics ( gender: female), biochemical indicators of liver failure (bilirubin, international normalized ratio), and histological features predict long-term survival; during follow-up, alcohol abstinence is a significant predictor of survival in both compensated and decompensated alcoholic liver disease patients. The presence and extent of bilirubin sludge, giant mitochondrial deficiency, and the degree of polymorphonuclear leukocyte infiltration have also been reported to be significantly associated with mortality in patients with alcoholic hepatitis; histologic scores of alcoholic hepatitis are highly accurate in predicting glucocorticoid efficacy and mortality. Alcoholic liver disease-associated malignancies should not be ignored. The results of a multicenter study from Japan showed that patients with liver cancer due to alcoholic liver disease were younger and had higher levels of liver fibrosis compared with non-alcoholic fatty liver disease, and the results of a Finnish survey including 1873 patients with alcoholic liver disease showed that the incidence of malignancies was significantly higher in patients with alcoholic liver disease than in the control population; among them, patients with severe alcoholic liver disease had hepatocellular liver cancer, upper gastrointestinal tract cancer, the The study also noted that the risk of malignant tumors was not found to be reduced in any of the patients with alcoholic liver disease investigated.