The fallopian tubes have the function of transporting sperm, picking up eggs and transporting fertilized eggs to the uterine cavity, so their patency is one of the essential conditions for conception. About 50% to 60% of infertile women are due to tubal factors. Organic lesions of the fallopian tubes such as inflammation, adhesions and obstruction prevent the sperm and egg from meeting in the lumen or affect the transport of the fertilized egg, thus causing infertility or ectopic pregnancy. Tubal infection is the main culprit of tubal obstruction. When tubal infection occurs, the narrowest part of the fallopian tube and the umbilical end of the tube can easily become adherent or completely occluded. The first site of tubal inflammation is often the lining of the fallopian tubes, causing swelling, interstitial edema, congestion and exudation. The mucosal epithelium of the fallopian tubes is detached, causing the mucosa to adhere to each other or to the umbilical ends of the fallopian tubes, leading to lumenal atresia and infertility. The cause of tubal infection is due to pathogenic infection. The main pathogens are Staphylococcus, Streptococcus, Escherichia coli, Gonococcus, Aspergillus, Pneumococcus, Chlamydia and Mycoplasma. The most likely time for infection to occur is after childbirth, miscarriage or menstruation. Damage to the abruptive surface of the birth canal and placenta caused during childbirth or miscarriage or the trauma of endometrial exfoliation during menstruation are ways for pathogens to infect the internal genitalia. Sometimes the infection is related to aseptic surgical procedures, such as IUD placement, curettage, tubal lavage, iodography, etc. Frequent sexual intercourse and sexual intercourse during menstruation can also cause infection and tubal infection. In a few cases, the infection is caused by the direct spread of inflammation from neighboring organs, such as appendicitis or infection from other parts of the body that spread through the bloodstream to the fallopian tubes.