Treatment of frozen gait includes medication, surgery, rehabilitation training and the application of assistive devices. Based on the response of patients with frozen gait to dopamine-like drugs, frozen gait can be classified into 3 types: drug-sensitive, drug-resistant, and drug-induced. Drugs with an evidence-based medical evidence classification of IA that are effective in the treatment of frozen gait include levodopa, dopamine receptor agonists, and monoamine oxidase B inhibitors. Some studies have shown that intravenous amantadine is effective in dopamine-resistant freezing gait. levo-3,4-dihydroxyphenylserine in combination with encatapone has also been shown to be effective in freezing gait that is ineffective against levodopa treatment in a study by Fukada et al. The use of methylphenidate, a CNS stimulant that effectively inhibits catecholamine reuptake and to some extent increases brain dopamine levels, is still controversial in the treatment of freezing gait. The reduced responsiveness of patients with advanced PD to medications and the prolonged “off” phase make the improvement of frozen gait with medications significantly less effective, and other appropriate treatments need to be sought. Surgical treatment of frozen gait includes deep brain stimulation in the thalamic nucleus, deep brain stimulation in the pontine nucleus, non-invasive repetitive transcranial magnetic stimulation, and transcranial direct current stimulation. To date, the therapeutic effect of STN-DBS on freezing gait is still uncertain, and Fasano et al. showed that STN-DBS reduced the frequency and duration of freezing gait episodes. STN-DBS significantly improved the freezing gait in the “off” phase compared to the freezing gait in the “on” phase. A prospective controlled study showed that STN-DBS reduced the frequency and severity of freezing episodes compared to continuous pharmacological treatment as early as 6 months after surgery and up to the 12th month of follow-up. STN-DBS can significantly improve the main motor symptoms of PD, but has been shown to induce impaired speech and dysarthria in some patients. There is growing evidence that rehabilitation is beneficial for the recovery of motor function in PD patients and facilitates the improvement of frozen gait. 1. Sensory cues: External audiovisual and proprioceptive cues can improve gait abnormalities in PD patients, even those who have not been treated with medication. Visual cues such as a stick placed on the ground or a striped line on the road can help PD patients overcome the onset of frozen gait. Many studies have shown that rhythmic auditory stimulation can be effective in improving gait disturbances in PD patients. PD patients with altered cognitive function may result in greater reliance on sensory information cues. 2. Physical therapy: Plate bicycle training can improve gait coordination and reduce the number of episodes of frozen gait. Two studies have shown that long-term robot-assisted mobility plank training can reduce the number of episodes of frozen gait and is a viable and effective means of rehabilitation. An open study suggests that RAS-based motor learning training may be useful for freezing gait. 3. Assistive devices: The main purpose is to provide additional support and visual and auditory information stimulation to the patient. A cane with a light source can provide visual cues and is easy to implement. Auditory cues can be delivered through wireless headphones and used in conjunction with a real-time gait monitoring system to achieve the integration of monitoring and lifting of frozen gait. Wheelchairs with power are helpful for patients who are unable to walk in advanced stages. The above treatments and rehabilitation methods focus on different aspects and are not effective for all patients, so it is important to individualize the selection of treatment measures.