Brain neuron cells work constantly like hard-working bees, but little energy is stored in the cell body, so when there is a sudden occlusion of the cerebral blood vessels and a disruption of blood flow, cerebral ischemia and hypoxia, neurological deficits, hemiparesis or coma will soon occur. This is how stroke develops, and its rate of disability and death is so high that it has risen to the top of the list of deaths from various diseases in China. Atherosclerosis, including cerebral atherosclerosis, has its beginnings in adolescence. Vascular endothelial damage begins before the age of 20, due to over-nutrition, obesity, smoking, alcoholism, emotional stress, hypertension and other adverse factors superimposed, resulting in endothelial damage after oxidative modification of low-density lipoprotein formation of foam cells invaded to the vascular endothelium, and the gradual formation of lipid streaks and lipid plaques, lipid plaques by the encapsulated lipid nuclei and outside the composition of the fibrous cap, the early fibrous cap is solid It is not easy to rupture, with the evolution of plaque, the fiber cap becomes thin, the lipid nucleus increases in size, forming a vulnerable plaque with “thin skin and big stuffing”, in the case of stress or weakness of the body, the circulating blood inflammatory changes, the blood viscosity increases, the platelet aggregation rate increases, which will lead to plaque rupture, thrombosis, leading to cerebral infarction, and the emergence of a serious situation. Understanding the mechanism of cerebral atherosclerosis and thrombosis, we should start from different links, and take corresponding preventive measures for different physiques at different ages. However, stroke occurs more often in middle-aged and elderly people, so prevention is especially important for high-risk groups. We generally categorize prevention into primary prevention and secondary prevention. Primary prevention is aimed at preventing strokes when they do not occur and when there are no symptoms. If there are already serious risk factors such as hypertension, diabetes, hyperlipidemia, smoking, poor lifestyle habits such as physical inactivity, hyperhomocysteinemia, etc., we should pay great attention to them, and give medication such as aspirin and statin to prevent them, and pay attention to a low-salt and low-fat diet, proper exercise, and control the risk factors, such as hypertension, hyperglycemia, hyperlipidemia, and smoking cessation. Secondary prevention: It refers to preventing the recurrence of cerebral infarction after a stroke has occurred. It has been reported that the recurrence rate of cerebral infarction reaches 30-40%, therefore, after suffering from cerebral infarction, one should be hospitalized immediately in the acute or hyper-acute stage, and be guided by specialists to actively rehabilitate the lost neurological functions, and on the other hand, take intensive measures to actively carry out secondary prevention, which mainly refers to double antiplatelet, intensive lipid-lowering, moderate antihypertensive, blood glucose control, smoking cessation and alcohol restriction, appropriate exercise, and comprehensive conditioning. Note: This is the principle of prevention that we emphasize, and the specific operation should choose individualized strengthening program.