21 Diagnostic basis, classification of symptoms and assessment of efficacy of addiction rash
Addiction rash is also known as bei (bei) lei (lei). It is characterized by itching of the body, and when scratched, erythematous elevations appear, shaped like beanpads, piling up into patches, occurring in indefinite places, appearing and disappearing, and leaving no trace after retreating. It is equivalent to urticaria.
21.1 Diagnosis based on Feng Jianqing, Department of Dermatology, Taicang Traditional Chinese Medicine Hospital
21.1.1 Sudden onset, the lesions are edematous plaques of different sizes and shapes with clear boundaries.
21.1.2 The rash starts and falls, itches intensely, is indefinite, and leaves no trace after retreating.
21.1.3 Some cases may have abdominal pain and diarrhea, or fever and arthralgia. Severe cases may have respiratory difficulties and even cause asphyxia.
21.1.4 Positive skin scratch test.
21.1.5 A rash that does not heal after more than three months or recurrent intermittent attacks is considered chronic addiction rash.
21.2 Classification of symptoms
21.2.1 Wind-heat offending surface: bright red, burning and severe itching. Accompanied by fever, chills, sore throat, and the rash is aggravated by heat. Thin white or thin yellow tongue coating and floating pulse.
21.2.2 Wind-cold surface: white rash, aggravated by wind-cold, decreases when warm, no thirst in the mouth. The tongue is pale, with white tongue coating and a floating tight pulse.
21.3.3 Blood deficiency and wind dryness: recurrent attacks, prolonged, aggravated in the afternoon or at night. With irritability, dry mouth, and heat in the hands and feet. Red tongue with little fluid and sunken pulse.
21.3 Evaluation of curative effect
21.3.1 Cured: The wind mass subsides, the clinical signs disappear, and no further occurrence.
21.3.2 Improvement: The wind masses recede 30% or the recurrence interval is prolonged after receding, and the itching and other symptoms are reduced.
21.3.3 Failure to heal: The wind mass and itching do not improve significantly, or the remission is less than 30%.
22 Diagnostic basis, classification of symptoms, and assessment of efficacy of earth wind sores
The skin disease is characterized by the appearance of a rash with a broken head, and the initial onset of disease. It is similar to papular urticaria.
22.1 Diagnostic basis
22.1.1 Mostly edematous red bumps with small hard blisters in the center, itching is intense, often with crusting. The rash often appears in batches, and it is recurrent and lingering.
22.1.2 It occurs mostly on the extensor surfaces of the extremities, abdomen, buttocks, etc.
22.1.3 Most often seen in infants and children, with onset in summer and autumn.
22.1.4 The disease can be triggered by insect bites, gastrointestinal dysfunction, intestinal parasitism, inappropriate diet, excessive consumption of sugar and animal protein, etc.
22.2 Classification of symptoms
22.2.1 Wind-heat offending the surface: wind mass-like diamond-shaped erythema with small papules or blisters in the center. Red tongue, thin white coating, floating pulse.
22.2.2 Damp heat in the stomach and intestines: erythema of wind masses with crusting and erosion. With abdominal distention and constipation. Slightly red tongue, white and greasy coating, smooth pulse.
22.3 Assessment of curative effect
22.3.1 Cured: All the self-conscious symptoms and skin lesions have disappeared.
22.3.2 Improvement: The papular eruption regresses by more than 30%, and the self-conscious symptoms are significantly reduced.
22.3.3 Not cured: no change in skin lesions and self-conscious symptoms, or less than 30% improvement.
23 Diagnostic basis, classification of symptoms and assessment of efficacy of drug toxicity
Drug toxicity refers to the skin rash caused by endowment intolerance and internal invasion of drug toxicity. It is equivalent to drug-related dermatitis.
23.1 Diagnostic basis
23.1.1 The lesions are mostly symmetrically distributed with widespread attacks. The shape varies, such as urticaria-like, measles-like, scarlet fever-like, erythema multiforme-like, or herpetic epidermolysis bullosa-like. The palmoplantar and oral mucosa are often more common.
23.1.2 There is a certain incubation period, the length of which is variable. It usually occurs within three weeks after drug administration.
23.1.3 The onset is rapid, with burning and itching, and may be accompanied by fever, lethargy, and other systemic symptoms. Severe cases may be accompanied by visceral damage.
23.1.4 History of drug use before the onset of the disease.
23.2 Classification of symptoms
23.2.1 Dampness and toxicity in the skin: erythema, blisters, or even vesicles and oozing, epidermal peeling. With severe itching, irritability, dry mouth, dry stool, yellow and red urine, or fever. The tongue is red, with thin white or yellow coating and smooth or counted pulse.
23.2.2 Heat toxin entering the camp: bright red or purplish red rash, or even purple spots and blood blisters, high fever and confusion, dry mouth and lips, thirst and inability to drink, dry stool and short red urine. The tongue is reddish, with little coating or mirrored tongue, and the pulse is torrential.
23.2.3 Qi and Yin deficiency: The rash subsides with low-grade fever, thirst, weakness, shortness of breath, dry stool, and yellow urine. Red tongue with little coating and fine pulse.
23.3 Assessment of curative effect
23.3.1 Cured: The rash subsides and the clinical signs disappear.
23.3.2 Improvement: The rash and clinical signs recede by more than 30%.
23.3.3 Not cured: The rash and signs are not relieved, or even aggravated.
24 Diagnostic basis, classification of symptoms, and assessment of efficacy of pemphigus
Pemphigus is caused by heart fire, spleen dampness, and wind-heat and summer dampness, which fumigate the skin. It is a herpetic skin disease in which the skin is covered with blisters as small as gravy or as large as chess pieces, and the skin is broken and dripping with fluid, and does not heal.
24.1 Diagnostic basis
24.1.1 Pemphigus vulgaris
24.1.1.1 The appearance of large, loose blisters on the skin with easily broken walls and vesicles that do not heal easily after breaking.
24.1.1.2 The lesions may be limited or spread throughout the body. Oral mucosal damage is common, or may occur first on the oral mucosa.
24.1.1.3 Positive for the Nisei sign.
24.1.1.4 The disease is most common in middle-aged people.
24.1.1.5 Histopathologic examination shows intraepidermal macules, commonly with loosening of spiny cells and formation of aspergilloma cells.
24.1.1.6 Direct immunofluorescence examination reveals IgG deposition between spicocytes with fishnet fluorescence.
24.1.2 Proliferative aspergillosis
24.1.2.1 Early lesions are similar to those of common aspergillosis, but papilloma-like proliferation is seen on the vesicular surface.
24.1.2.2 Preferably in the folds, or mucosal areas.
24.1.2.3 Positive for Ney’s sign.
24.1.2.4 Histopathological examination is the same as that of common aspergillosis in the early stages, and papilloma-like proliferation is seen later.
24.1.2.5 Direct immunofluorescence examination is the same as that of the common type.
24.1.3 Deciduous aspergillosis
24.1.3.1 The lesion is initially a shallow, flaccid blister that breaks easily and later appears as a large epidermal exfoliated vesicle covered with leafy crusts.
24.1.3.2 The rash tends to be generalized, but mucosal damage is rare or mild.
24.1.3.3 Positive Ney’s sign.
24.1.3.4 Histopathologic examination shows spine loosening in the granular layer and below, forming fissures and blisters.
24.1.3.5 Direct immunofluorescence examination is the same as the common type.
24.1.4 Erythematous aspergillosis
24.1.4.I Flaccid blisters on an erythematous base, or overlying seborrheic scabs.
24.1.4.2 Most often occurs on the cheeks, chest, back, or areas with high oil production. Mucosal damage is less common.
24.1.4.3 Positive for Ney’s sign.
24.1.4.4 Histopathological examination is the same as that of the deciduous type. Direct immunofluorescence examination is the same as for the common type of aspergillosis.
24.1.5 Pemphigus vulgaris
24.1.5.1 The lesions appear as tension blisters on the basis of normal skin or erythema, with thick, plump and tense walls that are not easily ruptured.
24.1.5.2 The lesions are more common in the flexural and crepitant areas of the extremities, and can also be generalized throughout the body, with self-induced pruritus. Mucosal damage is rare and not severe.
24.1.5.3 Negative for Ney’s sign, but positive for advanced blistering may occur.
24.1.5 The disease is most often seen in the elderly, but may also be seen in children.
24.1.5.5 Histopathological examination shows subepidermal tension blisters without aspergilloma cells.
24.1.5.6 Direct immunofluorescence examination shows linear IgG deposits along the basement membrane.
24.2 Classification of symptoms
24.2.1 Hot toxin: Rapid onset, rapid expansion or increase of blisters, bright red erosion. Body heat and thirst, dry stool and red urine. The tongue is red and vivid, with little or yellow coating, and the pulse is smooth or counted.
24.2.2 Burning heart fire: Redness of the oral erosion or sore surface, disturbed heart and thirst, short and red urine. The tongue is red, with yellow fur and counted pulse.
24.2.3 Damp-heat entrapment: large erosion surface or wet rotten flakes, thirst without desire to drink or nausea and vomiting. The tongue is red, the coating is yellow and greasy, and the pulse is smooth and counted.
24.2.4 Spleen deficiency with dampness: crusting, thicker and not easy to fall off, or tense herpes wall, flushed and indistinct. Lethargy and weakness, abdominal distension and loose stools. Pale and fat tongue, white and greasy coating, sluggish pulse.
24.2.5 Qi-yin injury: The disease has been in progress for a long time and no blisters appear. Lethargy and weakness, shortness of breath and lazy speech, or irritable heat in the five hearts. The tongue is light red, with little coating or peeling coating, and the pulse is sunken and thin.
24.3 Assessment of curative effect
24.3.1 Cured: The rash is all gone and no new rash recurs.
24.3.2 Improvement: the rash subsides more than 30%, with occasional new rash.
24.3.3 Not cured: The rash subsides by less than 30%, with new rashes occurring continuously.
25 Diagnostic basis, classification of symptoms, and assessment of efficacy of fire blisters
Fire red sores are herpetic skin diseases characterized by clusters of blisters, mostly arranged in a circular pattern, accompanied by erythema and unbearable itching. It is similar to herpes-like dermatitis.
25.1 Diagnostic basis
25.1.1 The lesions are mostly clusters of small blisters, often arranged in rings, with thick walls that are not easily ruptured. They occur mostly on top of a wind-blown mass and are intensely pruritic.
25.1.2 The lesions are usually found on the shoulders, buttocks, and extensor surfaces of the limbs. The mucous membranes are rarely involved. Pigmentation remains after the rash subsides.
25.1.3 The disease is most often seen in middle-aged people.
25.1.4 Negative for Ney’s sign.
25.1.5 Histopathological examination is subepidermal blistering.
25.1.6 Direct immunofluorescence examination shows granular deposition of IgA in the dermal papillae. A punctate fluorescence reaction is observed.
25.2 Classification of symptoms
25.2.1 Spleen deficiency with dampness: clusters of blisters with papules and severe itching like wind masses. Abdominal distension and dullness, loose stools, heavy limbs. Pale tongue, white coating, and smooth pulse.
25.2.2 Qi-yin injury: The disease lasts for a long time and no new herpes occur. Restlessness, laziness, weakness of the body, or five heartburn. The tongue is light red with little coating and the pulse is sunken and thin.
25.3 Assessment of therapeutic effect
25.3.1 Cured: All skin lesions disappear and no new rash reappears.
25.3.2 Improving: lesions recede more than 30%, with occasional new rash occurring.
25.3.3 Failure to heal: lesions recede less than 30%, with new rashes occurring continuously.
26 Diagnostic basis, classification of symptoms, and assessment of efficacy of dengdu sores
Dengdou sores are caused by a deficiency in the surface and a deficiency in the interior, with poisonous heat entering the blood. It is a skin disease characterized by large flushing of the skin and the appearance of clusters of pinhead- to corn-sized pustules, similar to herpes-like pustulosis.
26.1 Diagnostic basis
26.1.1 The lesions are small superficial pustules occurring on the basis of a large flush, which may fuse with each other to form a pus paste, a batch of dry crusts, and a new batch of pustules may appear or form a multi-ring. The rash is often preceded by a high fever.
26.1.2 Prevalent in the axillae, groin and other folds, can be generalized and can involve mucous membranes.
26.1.3 Acute attacks are often accompanied by high fever and chills.
26.1.4 It occurs mostly in the second trimester of pregnancy and may heal spontaneously after delivery.
26.1.5 Bacterial culture and blood culture of unbroken pustules are negative.
26.2 Classification of symptoms
26.2.1 Heat entering the blood: Cluster of pustules with flushed base. With high fever, chilliness, short red urine and dry stool. Red-red tongue, yellow or greasy coating, slippery or counted pulse.
26.2.2 Injury to both qi and yin: The disease has been in progress for a long time and no new pustules occur. The skin is light red, flaky, short of breath and weak, or the five hearts are troubled and hot. The tongue is light red, with little coating and fine pulse.
26.3 Assessment of curative effect
26.3.1 Cured: all skin lesions disappear, no new rash recurrence.
26.3.2 Improving: lesions recede more than 30%, no high fever.
26.3.3 Not cured: The occurrence of skin lesions is not controlled.
27 Diagnostic basis, classification of symptoms, and assessment of efficacy of erysipelas
Erythroderma is a systemic disease in which erythema resembling a butterfly often occurs on the face and may be accompanied by systemic lesions such as joint pain and internal organ damage. It is equivalent to lupus erythematosus.
27.1 Diagnostic basis
27.1.1 Systemic erysipelas
The diagnosis is made when four or more of the following items are present, either consecutively or simultaneously.
27.1.1.1 Butterfly erythema: raised or flat fixed erythema on the cheek. No skin lesions in the nasolabial area.
27.1.1.2 Discoid erythema: red elevated patches with adherent, keratinized scales and follicular plugs attached to the surface, with atrophic scarring visible in old damage.
27.1.1.3 History of photosensitivity or rash due to abnormal reaction to light on examination.
27.1.1.4 Stains in the mouth or nasopharynx, often painless.
27.1.1.5 There may be tenderness, swelling, or fluid accumulation involving two or more peripheral joints.
27.1.1.6 There is a history of definite chest pain or physical examination reveals pleural friction sounds or pleural effusion. or pericardial rubbing sounds on cardiac auscultation and pericardial effusion on laboratory tests.
27.1.1.7 Persistent proteinuria, 24-hour urine protein greater than 0.5 g and visible urine red blood cells, white blood cells, granules, and tubular
27.1.1.8 Exclusion of drugs or metabolic disorders such as uremia, ketonemia, electrolyte disorders, etc. with convulsions or psychiatric symptoms.
27.1.1.9 Blood tests: hemolytic anemia or leukocytes less than 4000/mm3 (4×109/liter); or lymphocytes less than 15% (1. 5×109/liter); or platelets less than 100,000/mm3 (100×109/liter).
27.1.1. 10 Immunological examination: positive lupus erythematosus cells or abnormal anti-dsDNA antibody titer or false positive SM antibody or syphilis serological reaction.
27.1.1. 11 Positive fluorescent antinuclear antibodies
27.1.2 Discoid erysipelas
The lesions are found on the cheeks, brow arches, auricles, lips, backs of hands and other areas of exposure, and appear as dark purplish-red infiltrative patches with a complex of filmy scales and some visible atrophy. The diagnosis is confirmed by a positive lupus band test, with liquefied basal cell degeneration, focal lymphocytic infiltration around the dermal vessels and adnexa on dermal pathology.
27.1.3 Subacute cutaneous lupus erythematosus: The lesions are mostly annular erythematous or polymorphic in nature. The pathological manifestations are the same as in 1.2. The systemic damage is mild and the antinuclear antibody is mostly positive.
27.2 Classification of symptoms
27.2.1 Hot and toxic: equivalent to the acute active stage of systemic erysipelas. The face is brightly colored with butterfly-shaped erythema and purple skin spots. It is accompanied by high fever, irritability and thirst, delirium, convulsions, joint and muscle pain, dry stools and short red urine. The tongue is red and vivid, with yellowish greasy coating and a flooded or fine pulse.
27.2.2 Injury to both Qi and Yin: Dark red rash. Accompanied by irregular fever or persistent low fever, fever in the hands and feet, distress and weakness, spontaneous sweating and night sweating, puffy red face, arthralgia, heel pain, scanty menstruation or amenorrhea. The tongue is red, the fur is thin, and the pulse is fine.
27.2.3 Spleen and kidney yang deficiency: puffiness of the face, puffiness of the eyelids and lower extremities, swelling and fullness in the chest, soreness and weakness of the waist and knees, heat in the face and coldness in the extremities, dryness of the mouth without thirst, scanty or closed urination. The tongue is pale and fat, with little coating and sunken pulse.
27.2.4 Spleen deficiency and liver exuberance: purple spots on the skin. Distention and fullness in the chest, abdominal distension and dullness, dizziness and headache, tinnitus and insomnia, irregular menstruation or amenorrhea. Purple tongue or petechiae, thin and stringent pulse.
27.2.5 Qi stagnation and blood stasis: Mostly seen in discoid restrictive and subacute cutaneous erysipelas. Erythema is dark and stagnant, with angular embolism and skin atrophy. With lethargy and weakness. The tongue is dark red with white coating or glossy tongue, and the pulse is sunken and thin.
27.3 Assessment of therapeutic efficacy
27.3.1 Systemic erysipelas
27.3.1.1 Evidence of efficacy: disappearance of symptoms and signs, significant decrease of antinuclear antibody titers and negative or low titers of anti-ds-DNA antibodies in laboratory tests, and remission can be maintained with continuous medication.
27.3.1.2 Improvement: Symptoms and signs largely disappear or decrease, laboratory indexes improve, and the disease can remain stable with continuous medication.
27.3.1.3 Not resolved: No improvement in symptoms and signs and laboratory tests.
27.3.2 Discoid erysipelas
27.3.2.1 Cured: The lesions disappear, the histopathological examination of dermal inflammation subsides, there is no liquefaction of basal cells, and the lupus band test is negative.
27.3.2.2 Progression: disappearance of most of the lesions and improvement of laboratory tests.
27.3.2.3 Failure to heal: No improvement in lesions or new rash, no change in physical signs and laboratory tests.
28 Diagnostic basis, classification of symptoms and assessment of efficacy of dermatomyositis
Dermatological paralysis is a skin disease with swelling and sclerosis of the skin and later atrophy. It may be confined to a certain area or may involve the whole body. It is similar to scleroderma.
28.1 Diagnostic basis
28.1.1 Systemic dermatophytosis
28.1.1.1 Initial edema of the fingers on the back of the hands and upper face, with symmetric diffuse puffy sclerosis of the skin, and advanced sclerosis of the skin and flexural atrophy of the fingers. Arterial spasm phenomenon in the extremities (Raynaud’s phenomenon), ulceration or scar formation in the uncinate of the fingers and toes. Multiple joint pain or swelling.
28.1.1.2 Pulmonary fibrosis on radiographs. dilatation and hypocontractility of the lower esophagus on barium radiography.
28.1.2 Localized dermatomalacia
28.1.2.1 Initially, there is a limited edematous plaque, followed by an ivory-colored sclerotic skin patch with a waxy sheen. It is surrounded by a pale red or purplish halo during the active phase, and skin atrophy appears in the late phase. Pathological tissue examination helps to confirm the diagnosis.
28.1.2.2 Pathologic biopsy of the skin on the extensor side of the forearm shows epidermal thinning, loss of epidermal protrusions, and swelling or fibrosis of dermal collagen fibers.
28.1.3 The patient is mostly female. There is mostly irregular fever, significant shortening of the tongue tie, mottled face, neck and palms, and multiple capillary dilation.
28.1.4 Increased sedimentation, positive rheumatoid factor, anti-Sc1-70 antibodies and autoantibodies such as anti-adhesion sites. X-rays show resorption of uncinate bones or soft tissue calcium deposits.
28.2 Classification of symptoms
28.2.1 Cold and damp obstruction: Most often seen in limited dermatomalacia. The skin is hard to the touch, waxy and shiny, and the hands are not able to squeeze it, with gradual atrophy. The tongue is pale or dark, with a thin white coating and a sunken or delayed pulse.
28.2.2 Spleen and kidney Yang deficiency: Most often seen in systemic dermatomyositis. Initially, the skin lesions are edematous and gradually become hard and atrophic. The patient feels weak, cold in the extremities, painful joints and even limited movement, bloating, loose stools, irregular menstruation or menopause. The tongue is pale, fat and tender or with tooth marks on the side, and the pulse is sunken and volatile.
28.3 Evaluation of therapeutic effect
28.3.1 Effective: More than 50% of the main symptoms such as skin sclerosis, arthrosis and arterial spasm of the extremities disappear, skin lesions become soft, pigmentation may remain or disappear, and pathological examination improves significantly.
28.3.2 Effective: The main symptoms disappear more than 30%, the pathological examination is improved, and the disease can be kept stable with continuous medication.
28.3.3 Not cured: No improvement or aggravation of the disease.
29 Diagnostic basis, classification of symptoms and assessment of efficacy of fox confusion disease
Fox confusion disease is a comprehensive skin disease characterized by erosion of the mouth, throat and pubic area and red eyes like the eyes of a turtledove. It is similar to leukoaraiosis syndrome.
29.1 Diagnostic basis
29.1.1 Repeated episodes of oral ulcers, or vulvar ulcers.
29.1.2 May be associated with melanoma (erythema nodosum), cyanosporin (subcutaneous thrombophlebitis), and a positive skin prick reaction. Or recurrent anterior chamber pus-accumulating iridocyclitis, chorioretinitis, and painful red and swollen joints, canker sore (appendicitis)-like abdominal pain, and black stool may occur in the eye.
29.1.3 It may be complicated by canker sores (epididymitis).
29.1.4 Some severe cases may develop central nervous system lesions, such as brainstem syndrome and meningoencephalitis syndrome, which may be complicated by occlusive vasculitis and aneurysm.
29.2 Classification of symptoms
29.2.1 Damp-heat toxic nodules: Mostly seen in the acute attack. Erythema nodosum of the lower extremities, symptoms include high fever, disturbed sweating, sores on the mouth and tongue, burning pain, aching joints, stuffiness and distension in the chest, bitterness in the mouth and dryness in the throat, and yellow and thick banding in women. The tongue is red, the coating is yellow, and the pulse is stringent and slippery.
29.2.2 Liver and kidney yin deficiency:, mouth, eye and vulvar ulcers are light and heavy at times, with recurrent attacks, lingering low fever, heat in the hands and feet, dizziness, dry mouth and throat, seminal emission and night sweating, irregular menstruation, soreness and weakness of the waist and knees. Red tongue with little fluid or cracked tongue or glossy tongue with fine pulse.
29.2.3 Spleen and kidney yang deficiency: recurrent oral and vulvar ulcers, aggravated by cold. The duration of the disease is long, with generalized weakness, little breath and lazy speech, lack of warmth in the hands and feet, loss of appetite, fear of cold, swelling of the lower limbs, loose stools, seminal emission and impotence, and menstrual irregularities. The tongue is pale, the fur is white, and the pulse is sunken and thin.
29.3 Evaluation of therapeutic effect
29.3.1 Significant effect: The main symptoms such as ulcers in the mouth, eyes, vulva and skin nodules mostly disappear, the acupuncture reaction is negative, and the condition is stable.
29.3.2 Improvement: The main symptoms are obviously reduced, and the condition can be kept stable with continuous medication.
29.3.3 Not cured: The condition does not improve or worsens.
30 Diagnostic basis, classification of symptoms, and assessment of efficacy of catarrh
Cat’s eye sores are named after the rash that resembles the eyes of a cat. It occurs most often in spring and autumn and is most common on the hands and feet, and may involve the mouth and pubic area. It is comparable to erythema multiforme.
30.1 Diagnostic basis
30.1.1 The skin lesion starts as an erythematous spot, slightly above the skin surface, and later blisters appear in the center, about the size of lentils or finger caps. Initially bright red, gradually become dark red or dark purple-red, can be fused with each other, the erythema may fade in the center, forming a ring, or overlapping blisters such as iridescence. If the blisters are bloody, they are called hemorrhagic catarrh. It is painful and slightly itchy.
30.1.2 The onset of the disease is rapid, and the rash may be preceded by general malaise and other prodromal symptoms, often accompanied by pharyngitis, tonsillitis, and arthritis.
30.1.3 The rash is usually symmetrical on the finger edges, palms, forearms, dorsum of the feet, calves, face and neck. In severe cases, mucous membranes may be involved.
30.1.4 The incidence is more frequent in young women, and spring and autumn are the seasons.
30.1.5 Histopathological examination: epidermal cells are edematous, and subepidermal blister formation can be seen if exudation is obvious; dermis is edematous, small blood vessels are dilated, and inflammatory cells are infiltrated around. The early stage is neutrophilic and eosinophilic cells, the late stage is lymphocytes and histiocytes, and the collagen fibers are obviously swollen.
30.2 Classification of symptoms
30.2.1 Damp-heat encrustation: Rapid onset, bright red skin lesions with obvious central blisters. Fever, sore throat, dry mouth, joint pain, dry stool, yellow urine. The tongue is red, with white or slightly yellow coating, and the pulse is smooth or slightly counted.
30.2.2 Cold-damp obstruction: Dark red rash, aggravated by cold. Heavy lower limbs, painful joints, clear and long urine. Pale tongue, white fur, sunken or slow pulse.
30.3 Assessment of curative effect
30.3.1 Cured: All skin lesions disappear and self-conscious symptoms disappear.
30.3.2 Improvement: lesions fade more than 30%, self-conscious symptoms significantly reduced.
30.3.3 Not healed: lesions recede less than 30%, and new rash still occurs.