11 Diagnostic basis, classification of symptoms, and assessment of efficacy of scabies
Scabies is caused by scabies infestation of the skin. It is an infectious skin disease characterized by blisters and tunnels on the skin of the fingers, wrists, umbilicus, and pubic area, and itching at night.
11.1 Diagnostic basis
11.1.1 The lesions show scattered distribution of light red pinhead to chestnut-sized papules, papules and scabies tunnels. Small maroon nodules are sometimes seen on the pubic area. Itchiness is felt, especially at night.
11.1.2 It occurs between the fingers, wrists, elbow fossa, anterior axillary margin, inframammary, periaqueductal, inguinal and other folds, and can be generalized all over the body, except in children, where it rarely occurs on the head and face. Feng Jianqing, Department of Dermatology, Taicang City Hospital of Traditional Chinese Medicine
11.1.3 History of close contact with scabies patients.
11.1.4 Scabies can be detected in the skin lesions.
11.2 Classification of symptoms
11.2.1 Wind-heat in the skin: few blisters, many papules, thick walls, few waves, scratching and scabbing, itching, dry and thick skin for a long time, red tongue, thin coating, floating or slippery pulse.
11.2.2 Damp-heat poisonous gathering: skin blisters, papular rash flooding, thin-walled liquid, broken flow of fatty water, immersion wet rot. Or pustules stacked, or red filaments, swelling and pain in the nucleus of the family. The tongue is red, the coating is yellow and greasy, and the pulse is smooth.
11.2.3 Insect poisonous nodules: Isolated brownish-red nodules occur in the penis and scrotum, which itch from time to time and are difficult to dissipate for a long time. Pale tongue, white fur, slippery pulse.
11.3 Evaluation of curative effect
11.3.1 Cured: complete disappearance of symptoms and signs, more than three weeks, no recurrence.
11.3.2 Improvement: After the end of treatment, the itching is significantly reduced and the rash subsides by more than 60%.
11.3. 3 Not cured: No gradual resolution of symptoms and signs, or the rash subsides by less than 30%.
12 Diagnostic basis, classification of symptoms, and assessment of efficacy of snakebite
Snakebite is caused by dampness and heat in the liver and spleen, and by the presence of evil toxins. It is a skin disease characterized by clusters of watery sores distributed in bands along one side of the body, arranged like snakes, and with severe pain. It is comparable to herpes zoster.
12.1 Diagnostic basis
12.1.1 The lesions are mostly green bean-sized blisters in clusters with tense walls and red bases, often distributed unilaterally and arranged in bands. In severe cases, the lesions may appear hemorrhagic or gangrenous. Those with lesions on the head and face tend to have more severe disease.
12.1.2 The rash is often preceded by a tingling or burning sensation in the skin and may be accompanied by mild discomfort and fever in the periphery.
12.1.3 Conscious pain is obvious and may be severe and unbearable or may remain after the rash subsides.
12.2 Classification of symptoms
12.2.1 Liver meridian heat: bright red lesions, tense herpes wall, burning and stinging pain, bitter mouth and dry throat, irritability, dry stool or yellow urine. The tongue is red, the tongue coating is yellow or yellow thick, and the pulse is smooth and slippery. “
12.2.2 Spleen deficiency with dampness: lighter color, loose herpes wall, no thirst, little food and abdominal distension, loose stools at times, light tongue, white or white greasy tongue coating, sluggish or slippery pulse.
12.2.3 Qi stagnation and blood stasis: local pain persists after the rash subsides. The tongue is dark, with white coating and thin pulse.
12.3 Assessment of curative effect
12.3.1 Cured: The rash subsides, clinical signs disappear, and there is no pain sequelae.
12.3.2 Improvement: the rash subsides about 30%, and the pain is significantly reduced.
12.3.3 Not cured: The rash subsides by less than 30%, and there is still pain.
13 Diagnostic basis, classification of symptoms, and assessment of efficacy of heat sores
Most heat sores are caused by internal heat. It is characterized by clusters of blisters at the junction of the skin and mucous membranes, and itching and pain. It is comparable to herpes simplex.
13.1 Diagnostic basis
13.1.1 The lesions are red in color, with clusters of papules, blisters and crusts, and vesicles that break down and become itchy and painful.
13.1.2 Often occurs after fever or when resistance is low. It is easy to recur.
13.1.3 The lesions are usually found on the lips, skin and mucous membrane junction, and around the nostrils, cheeks and external genitalia.
13.2 Classification of symptoms
13.2.1 Heat in the lungs and stomach: small clusters of blisters, burning and itching. Mild peripheral discomfort, distraction and depression, dry stools, yellow urine. Red tongue with yellow fur and string pulse.
13.2.2 Yin deficiency internal heat: intermittent attacks, dry mouth and lips, slight heat in the afternoon. Red tongue, thin coating and fine pulse.
13.3 Assessment of curative effect
13.3.1 Cured: All skin lesions fade away, with light brown pigmentation spots remaining.
13.3.2 Improved: lesions dry, crust, or recede more than 30%.
13.3.3 Not healed: no change in lesions or less than 30% remission.
14 Basis of assertion, classification of symptoms and assessment of efficacy of wart
A wart is a small, flat, slightly elevated wart that occurs on the face or back of the hand. It is comparable to a flat wart.
14.1 Diagnostic basis
14.1.1 The lesion is a rice-grain to sorghum-sized flat papule with a smooth, isolated, scattered, light yellowish-brown or normal skin color, or slightly itchy.
14.1.2 Mostly on exposed areas, such as the face and back of the hands.
14.1.3 Characterized by auto-inoculation. Isomorphic reactions may be seen.
14.1.4 Prevalent in adolescents.
14.1.5 Histopathological examination: hypertrophy of the epidermal spiny layer, papillomatous hyperplasia and hyperkeratosis with incomplete keratinization. There are vacuolated cells in the upper spiny layer and granular layer with deep-stained, basophilic nuclei.
14.2 Classification of symptoms
14.2.1 Heat toxin accumulation: The rash is light red, with a large number of rashes, accompanied by dry mouth without desire to drink, body heat, irregular bowel movements and yellow urine. Red tongue, white or greasy coating, smooth pulse.
14.2.2 Heat-embedded stasis: longer duration of illness, with yellowish-brown or dark red rash, may have irritable heat. The tongue is dark red, the fur is thin and white, and the pulse is sluggish.
14.3 Assessment of curative effect
14.3.1 Cured: The skin lesions recede and no new rash appears.
14.3.2 Improvement: The rash is flatter than before, receding by more than 30% or with individual new rash appearing.
14.3.3 not healed: no change in the rash or less than 30% remission.
15 warts of the diagnostic basis, the classification of evidence, efficacy assessment
The warts are good in the hands, feet, scalp, as large as soybeans, rough and hard, the surface is spiky, equivalent to common warts.
15.1 Diagnostic basis
15.1.1 The lesions are hemispherical papular nodules of corn to soy size with rough and uneven surface like spines.
15.1.2 They occur on the back of the hands, toes, and edges of the feet.
15.1.3 Histopathological examination: marked surface keratinization and hypertrophy of the spiny layer, formation of a network of vacuoles in the upper epidermis, and papilloma-like hyperplasia.
15.2 Classification of symptoms
15.2.1 Wind-heat and blood dryness: nodules like beans, hard and rough, yellow or red in color. Red tongue, thin coating, and stringy pulse.
15.2.2 Damp-heat and blood stasis: loose nodules, gray or brown in color. Dark red tongue, thin white fur, thin pulse.
15.3 Evaluation of therapeutic effect
Same as for flat wart.
16 Diagnostic basis, classification of symptoms and assessment of efficacy of rat’s breast
The most important thing is that the wart is a little bit of a wart. It is a good idea to have a good look at the product.
16.1 Diagnostic basis
16.1.1 The lesions are hemispherical papules of corn to green bean size, milky white or normal, glossy surface, central umbilical fossa, isolated and scattered. When punctured, white powdery vesicles can be extruded. It is slightly itchy.
16.1.2 Prevalent on the trunk and extremities. –
16.1.3 Most commonly seen in children and young adults. It is contagious by contact.
16.2 Classification of symptoms
16.2.1 Wind-heat skin: shiny papules, slightly itchy, painful when scratched, slightly red all around. Red tongue, thin coating, thin pulse.
16.2.2 Damp-heat inclusion: the papule is itchy and scratches with juice, or has scratch marks, and can squeeze out powder-like white bodies after breaking. The tongue is red, the coating is thin and greasy, and the pulse is moist.
16.3 Evaluation of therapeutic effect
Same as for flat wart.
17 Diagnostic basis, classification of symptoms and assessment of efficacy of corns
Corns are caused by long-term compression or pressure on the foot (or occasionally on the hand), with the roots sunken into the flesh and hard nodules on top, resembling chicken eyes.
17.1 Diagnostic basis
17.1.1 The lesions are pea-sized, yellowish, cone-shaped nodules, firm, slightly above the skin surface, with smooth surface and obvious skin lines.
17.1.2 Prevalent in friction and pressure areas, mostly on the soles of the feet, between the toes, etc., with obvious pressure pain.
17.1.3 Footwear discomfort, prolonged friction and pressure, foot deformity, long-term walkers are prone to this disease.
17.2 Classification of symptoms
17.2.1 Phlegm-damp condensation: conical hard nodules on the surface, grayish yellow or waxy yellow, painful when pressed. Thin white tongue coating and slippery pulse.
17.2.2 Damp-heat toxic agglomeration: slightly red around the nodule, slightly swollen and painful under pressure. The tongue is red, the coating is thin, and the pulse is slightly counted.
17.3 Assessment of curative effect
17.3.1 Cured: The skin lesions recede.
17.3.2 Improving: lesions recede more than 30% and pressure pain is reduced.
17.3.3 Not healed: no change in damage or less than 30% regression.
18 Diagnostic basis, classification of symptoms and assessment of efficacy of callus
Callus is caused by friction and pressure on the hands and feet for a long time, and is characterized by thick, astringent, round and short skin like a callus.
18.1 Diagnostic basis
18.1.1 The skin lesion is a waxy yellow limited flat plaque. The central part is thickest and the marginal damage is thinner. There may be mild pressure pain.
18.1.2 The lesions are found in the palmoplantar area and other areas susceptible to pressure and friction.
18.2 Classification of symptoms
18.2.1 Phlegm and stasis: thick skin lumps round and short like calluses, waxy yellow, or with pressure pain. Red tongue, thin coating, slippery pulse.
18.2.2 Phlegm stagnation and poisonous stagnation: swollen lumps with purple-red surroundings and obvious pressure pain. Red tongue, thin coating, and faint pulse.
18.3 Evaluation of curative effect
18.3. 1 Cured: flattened thickened plaque subsides.
18.3.2 Improving: flat thickened plaques become thinner, more than 30% improvement, and pressure pain is obviously reduced.
18.3.3 Not cured: No change in lesions or less than 30% regression.
19 Diagnostic basis, classification of symptoms, and assessment of efficacy of eczema
Damp sores are caused by intolerance of endowment and the presence of wind-damp heat in the skin. The rash is in various forms, and the hair is not localized, so it is easy to wet and rot the itchy exudative skin disease. It is equivalent to eczema.
19.1 Diagnostic basis
19.1. 1 Acute eczema
19.1.1.1 The lesions are polymorphic, such as flushing, papules, blisters, vesicles, oozing, scabs, and flaking, and often several forms exist simultaneously.
19.1.1.2 The onset is rapid, with burning and intense pruritus.
19.1.1.3 The lesions are often symmetrically distributed, with the head, face, distal extremities, and scrotum being the most common sites. It may be generalized to the whole body.
19.1.1.4 The lesions may develop into subacute or chronic eczema, sometimes mild and sometimes severe, and repeatedly do not heal.
19.1.2 Subacute eczema: The lesions exude less and are dominated by papules, papules, crusts, and scales. There is a mildly vesicular surface with a darker red color. Mild infiltration and intense pruritus may also be seen.
19.1.3 Chronic eczema: mostly confined to one site, with clear boundaries, marked hypertrophic infiltrates, rough surface, or tinea-like changes, maroon or brown in color, often accompanied by papules, scabs, and scratches. Tendency to moist changes, often recurrent, sometimes light and sometimes heavy, with paroxysmal pruritus.
19.2 Classification of symptoms
19.2.1 Damp-heat infiltration: rapid onset, flushed and burning lesions, itching without rest, oozing fluid and juice. It is accompanied by body heat, irritability and thirst, dry stool and short red urine. The tongue is red, with thin white or yellow coating and smooth or counted pulse.
19.2.2 Spleen deficiency with dampness: The onset is slow, with flushed, itchy skin lesions, vesicles and oozing after scratching, with visible scales. It is accompanied by poor appetite, fatigue, abdominal distension and loose stools. The tongue is light and fat, with white or greasy coating and slow pulse.
19.2.3 Blood deficiency and wind dryness: The disease is prolonged, with dark lesions or pigmentation, severe itching, or rough and thick skin lesions. With dry mouth and no desire to drink, poor appetite and abdominal distension. The tongue is pale, the coating is free, and the pulse is thin and stringent.
19.3 Assessment of curative effect
19.3.1 Cured: The skin lesions subside.
19.3.2 Improvement: Skin lesions recede by more than 30%.
19.3.3 Not cured: lesions recede by less than 30%.
20 Diagnostic basis, classification of symptoms and assessment of efficacy of four curved winds
Four-bending wind is caused by endowment intolerance or spleen deficiency and dampness. It is a chronic skin disease characterized by dry and thick skin, scratching and itching. It is equivalent to atopic dermatitis.
20.1 Diagnostic basis
20.1.1 The lesions are characterized by dry, rough, hypertrophic mossiness, and may have acute or subacute dermatitis-like episodes. Self-conscious severe itching.
20.1.2 The lesions are usually found on the flexion side of the elbow and knee joints, but also on the extension side of the lower leg, the face and neck, and around the mouth.
20.1.3 There may be a history of infantile eczema with recurrent attacks that do not heal.
20.1.4 There is a genetic predisposition to allergies, with a family or personal history of asthma and addiction rash.
20.1.5 Elevated serum IgE and elevated eosinophils in the blood.
20.2 Classification of symptoms
20.2.1 Blood deficiency and wind dryness: dry and hypertrophic skin, itching and scratching with blood scabs. Abdominal distension after eating, constipation or loose stools. Light and fat tongue, white coating, smooth pulse.
20.2.2 Wind-damp skin: flushed skin, intense itching, scratching can be erosive and oozing. With tiredness and loose stools. The tongue is pale, the coating is thin and greasy, and the pulse is slippery.
20.3 Evaluation of therapeutic effect
20.3.1 Cured: The rash subsides, or there is pigmentation or hypopigmentation left.
20.3.2 Improvement: thinning and lightening of skin lesions, more than 30% remission, and reduction of itching.
20.3.3 Not cured: lesions recede less than 30%.