From the anatomical point of view, the muscles with high tension or easy to produce spasm are mostly in the inner posterior side of the lower leg and the bottom of the foot, such as the posterior tibialis, long toe flexor, [toe flexor, flounder and earthworm muscles, short toe flexor, etc., while the antagonistic muscles, especially the anterior tibialis, are weak. The antagonistic muscles, especially the anterior tibial muscles, are weak. Therefore, post-stroke foot pronation and ptosis are manifested to different degrees at the beginning of the disease. Therefore, there is a need to design a reasonable and targeted treatment for pronation and ptosis as early as possible. The main feature of post-stroke pronation and ptosis is the increased tone of the plantar flexors and the relatively low tone of the dorsiflexors. The dorsiflexors include the tibialis anterior, toe extensors and bunion extensors, and the extensor digitorum longus and peroneus brevis. If the dorsiflexors and extensor digitorum are targeted to excite and increase the muscle strength and tone, the inversion and plantar flexors are inhibited and antagonized, thus regulating the balance of muscle tone. Modern medical research has confirmed that the root cause of limb paralysis due to stroke is organic damage to the brain parenchyma, which causes the loss of adjustment of the senior center of the detrusor reflex. Inversion of the foot is caused by paralysis of the lateral calf muscle group and spastic pulling of the tibialis anterior muscle. Spasticity is a motor disorder characterized by an increase in the speed of the tonic retractor reflex due to abnormal sensory afferent nerve input to the crural medulla. Positive signs include excessive deep tendon reflexes, flexor spasm, abnormal spastic tone, and abnormal co-contraction. Negative signs include paralysis, joint band movements, lack of flexibility and fatigue. Spastic foot drop and pronation following hemiplegia have serious negative consequences for the patient, including spastic episodes, limitation of movement, pain, joint contractures, and functional impairment. The facilitation technique is the core technique of hemiplegia rehabilitation in the world in recent years. Traditional acupuncture treatment is also a facilitation technique, and the acupuncture points are mostly densely populated by various receptors, but because of the manual stimulation, the muscle contraction cannot produce proprioceptive impulses. 3, produce symptoms, stroke patients with foot drop, foot inversion deformity so that the patient’s foot contact with the ground is poor, the center of gravity has difficulty moving forward, stride and step frequency, step speed is reduced, when the patient moves forward to produce the typical toe dragging affect walking. 4, abnormal tone theory, stroke hemiplegia foot inversion is caused by increased tone of the extensor muscles of the lower limb and imbalance of the medial and lateral muscles of the limb, with the medial side of the lower limb being the spastic dominant side and the lateral side being the inferior side. The inversion of the foot is caused by spasm of the posterior tibial muscle, and the high tone of the foot directly affects the recovery of the motor function of the lower limb, which is not conducive to the improvement of walking ability and puts the patient at high risk of falling. Therefore, it is necessary to reduce foot spasm. The treatment of foot entropion due to lower limb spasticity after stroke is multidisciplinary. It occurs mainly due to medial tension and lateral retardation of the limb. The stroke patient is bedridden for a long time, the joint is braked, the ligaments are not stretched and lose their elasticity, and the muscles spasm, thus changing the biomechanics of the joint between the tibia and the heel bone, and the balance of the bone gap, the bone and the related muscles and ligaments are out of balance, resulting in inversion of the foot. This is an abnormal movement pattern, and blind and untargeted muscle strengthening training treatment will reinforce this primitive movement pattern and consequently prevent the establishment of advanced, normal movement patterns.