When a patient presents with the above clinical features and gastric torsion is suspected, x-ray examination can often help confirm the diagnosis. For acute gastric torsion, the diagnosis is mostly uneventful as long as the disease can be thought of. If a gastric tube is inserted to confirm the diagnosis, it should be inserted slowly and not forcibly to avoid damage or perforation of the gastric wall. Chronic gastric torsion is difficult to diagnose clinically because it is not completely obstructed and its symptoms are nonspecific. In adults, most gastric torsions have anatomic factors and are triggered by different triggers. The normal position of the stomach depends mainly on the fixation of the lower esophagus and the pylorus. The hepatogastric and gastrocolic ligaments and the gastrosplenic ligaments also play a role in fixing the large and small curves of the stomach. Larger esophageal hiatal hernia, septal hernia, septal bulge and excessive laxity of the lateral peritoneum of the descending duodenum make the lower end of the esophagus and the pylorus at the esophageal hiatal hernia not easily fixed. In addition, gastric prolapse and ligaments on the side of the greater and lesser curvatures of the stomach are lax or too long, which are anatomical factors in the development of gastric torsion. Acute gastric dilatation, acute colonic distention, overeating, violent vomiting and retroperitoneum can be the driving force for sudden changes in the position of the stomach, so they are often the precipitating factors for acute gastric torsion. Inflammation and adhesions around the stomach can pull the stomach wall and fix it in an abnormal position and cause torsion, and these lesions are often the cause of chronic gastric torsion.