The symptoms of bilateral basal ganglia cerebral infarction are not too serious clinically. Bilateral basal ganglia cerebral infarction is mainly caused by atherosclerosis of cerebral arteries, resulting in small vascular lesions in the brain, which are usually related to vascular sclerosis caused by long-term hypertension, hyperglycemia and hyperlipidemia. Patients may have clinical symptoms such as unilateral numbness in the limbs or mild unfavorable limb function. However, if the patient has more small lesions of bilateral infarcts in the basal ganglia area, forming a luminal state, it may also lead to a decrease in cognitive function. There are also symptoms of pseudobulbar palsy, choking and coughing when eating or drinking, and patients may also have a slow gait when walking, strong crying and strong laughing. Prevention of infarcts in the bilateral basal ganglia area of the brain is very important, and it is important to control the underlying disease and take oral aspirin to prevent the increase of luminal infarcts by antiplatelet aggregation.