Physical reactive rhinitis, also known as vasomotor rhinitis, can be divided into three clinical types, with some patients often having specific nasal reactions to certain rational stimuli. For example, episodes of sneezing, accompanied by more watery nasal discharge, occur whenever there is exposure to cold air, sudden temperature changes, humidity, etc. Patients can often clearly state the trigger. Patients can often clearly state the trigger for the onset. This type of rhinitis may also be a reverse hyperactivity rhinitis. Vasomotor rhinitis is a hyperreactive rhinopathy caused by an imbalance in the neuroendocrine regulation of the vascular and glandular functions of the nasal mucosa. The pathological mechanism of this disease is complex and many aspects are still not well understood, which makes the exact clinical diagnosis and effective treatment difficult. There are no significant gender differences in the onset of the disease, and vasomotor rhinitis rarely occurs in children. Goldman (1987) classified vasomotor rhinitis into three clinical types based on pathogenic factors, and this typing has some significance for diagnosis and treatment. （Some patients often have specific nasal reactions to certain rational stimuli. For example, episodes of sneezing with more watery nasal discharge may occur upon exposure to cold air, sudden temperature changes, humidity, etc. Patients can often clearly state the trigger for the onset. This type may also belong to reverse hyperactive rhinitis. (ii) Psychogenic reaction type Repeated stimuli such as mental tension, fear or resentment, frustration, etc., cause the patient’s nasal reaction. The contradiction between the changing content and accelerated pace of modern neurological life and traditional concepts has increased the number of such patients. (iii) Idiopathic reaction type Patients of this type often cannot find any suspected triggering factors and account for the majority of cases of vasomotor rhinitis. Patients often have watery nasal discharge, edema of the nasal mucosa, and sometimes mucosal polyps or nasal polyps. Endocrine dysfunction may be one of its triggers. According to the clinical characteristics, it can be divided into two types: 1. Nasal congestion type The symptoms of this type are mainly nasal congestion, which is mostly intermittent. Some patients have severe nasal congestion in the morning, which is reduced or disappears during the day. Some patients also get worse every night, often accompanied by alternating nasal congestion with changes in body position. If polypoid changes or nasal polyps occur in the nasal mucosa, there may be persistent nasal congestion of varying degrees. Sneezing occurs from time to time, but to a lesser extent. The nose may be relieved briefly after sneezing. Patients are often unusually sensitive to changes in climate and environmental temperature. 2. Nasal overflow type The main symptom is increased watery nasal discharge, mostly accompanied by episodes of sneezing. The onset of the disease is often for several days in a row, and several handkerchiefs are changed or a lot of toilet paper is used every day. Itching in the nose, but rarely with conjunctival involvement and itchy eyes. The symptoms can be relieved or disappear after a few days or weeks, and can develop again after a certain interval under the action of certain triggers. This type is more common in women aged 20 to 40 years old, and the mental type is more unstable. Other symptoms, there are still symptoms such as loss of smell and dizziness caused by continuous mucosal swelling and congestion and edema. There is no constant change in the color of the nasal mucosa on rhinoscopy. There is a dark red color produced by congestion, or a light blue color produced by dilated volume vessels, or a pale white color produced by mucosal edema. In some cases, one side of the nasal mucosa is congested and dark red, while the other side is pale and edematous. Enlarged turbinates generally respond well to ephedrine contraction, but those with long duration of disease or those who repeatedly use “nasal drip” have poor contraction response. Those with long duration of disease may show mucosal edema and polypoid changes. Postnasal microscopy may reveal enlarged and edematous posterior inferior turbinates.