Inability to speak after brain hemorrhage is usually a result of damage to the language center and can only be facilitated by follow-up treatment, such as medication and rehabilitation, to restore the patient’s language function. If a patient cannot speak after brain hemorrhage, it should be called post-stroke cognitive impairment. Cholinesterase inhibitors, such as donepezil, galantamine hydrobromide, and carbamate bitartrate, can be taken to promote the recovery of brain cells. NMDA receptor antagonists, such as memantine, can also be taken. You can also take brain cell resuscitators, such as nicergoline and sodium cytarabine, to promote the recovery of brain cells. At the same time, rehabilitation can be actively carried out to help patients recover with various physical means, such as language function recovery is learned from scratch, taught word by word like an elementary school student, learning to speak. Treatment can also be supplemented with acupuncture, herbal medicine, hyperbaric oxygen, and electrical stimulation. Brain hemorrhage cannot speak because the hemorrhage destroys the language center, which is divided into two, one is the motor language center and the other is the sensory language center. Disruption of the motor speech center results in motor aphasia, where the patient can hear but not speak. With disruption of the sensory language center, the patient can speak but cannot understand what is being said. If the amount of brain hemorrhage is large, both are damaged and the patient has mixed aphasia, in which the patient can neither understand nor speak.