IgA nephritis cannot be cured at present, but the aggravation of the disease can be delayed through active treatment. The pathogenesis of IgA nephritis is still unclear, mainly due to the abnormal immune function in the patient’s body, autoantibodies and immune complexes deposited in the glomeruli lead to inflammation. Currently there is no cure, but kidney damage can be reduced and delayed by regular monitoring, drug therapy, etc. Clinical manifestations, renal pathological changes and prognosis of patients with IgA nephritis vary greatly, and those with milder conditions only need regular monitoring of urinary protein and renal function. IgA nephritis treatment is mainly to control the patient’s proteinuria, such as the use of angiotensin-converting enzyme inhibitors such as captopril; if necessary, can be clarified under the pathology of the condition of the application of glucocorticoids and immunosuppressant treatment as prescribed by the doctor, such as prednisone, cyclophosphamide and so on. IgA nephritis patients should go to the hospital in time to improve the examination, under the guidance of the doctor active treatment, so as not to delay the condition.