Clinical manifestations of cardiogenic cirrhosis. In patients with congestive heart failure with passive hepatic stasis, most of the signs and symptoms are due to severe cardiac failure, while the manifestations of hepatic involvement are secondary. Mild right upper abdominal discomfort may be present, and 10-20% have jaundice. Physical examination may have congestive heart failure manifestations, including jugular vein rage, positive hepatic jugular venous reflux sign, most patients with hepatomegaly, a small number of patients can be highly enlarged, 50% of the patients more than 5cm below the costal margins, secondary tricuspid valve insufficiency can be palpable distended liver pulsation, with the formation of hepatic fibrosis the liver can be retracted to normal, 15% of the patients with ascites, 25% of the patients with splenomegaly. What are the easily confused symptoms? 1, enlarged liver Under normal circumstances, the liver in the upper right side of the abdominal cavity, in the rib cage surrounded by, and under the dome of the diaphragm, usually in the right side of the ribs can not be touched. If the liver can be touched 1.5 centimeters or more than 1.5 centimeters below the right rib when breathing calmly, it suggests that the right lobe of the liver is enlarged. In the mid-epigastric region, the liver should be within the upper 1/3 of the line between the xiphoid process and the umbilicus; if it is beyond that, it suggests enlargement of the left lobe of the liver. This is the initial diagnosis of hepatomegaly. There can be many reasons for hepatomegaly, which can be caused by hepatitis, cirrhosis, portal hypertension or other organs after organic or functional changes. 2, lifting-like beat refers to the heart slow, strong beat, can make the tip of the finger raised and continued until the beginning of the second heart sound, and at the same time the range of apical beat also increased, for the left ventricular hypertrophy symptoms. Systolic lifting-like beats at the left lower sternal border are a reliable indicator of right ventricular hypertrophy. 3, reverse beat When the heart contracts, the anterior wall of the left ventricle hits the chest wall of the precordial area in the early contraction, making the corresponding part of the rib question tissue outward beat, known as apical beat. After left ventricular myocardial infarction, the whole layer of myocardial necrosis of the ventricular wall. In about 10-38% of cases, the necrotic myocardium is gradually replaced by fibrous scar tissue, forming a ventricular wall tumor. The thin layer of the ventricular wall in the lesion area bulges outward, and the heart loses mobility or exhibits paradoxical motion during contraction. The evolution of coronary artery obstruction, myocardial infarction, myocardial fibrosis and left ventricular ventricular wall tumors was well recognized as early as 1881.