Temporal arteritis, also known as giant cell arteritis, occurs in women over 50 years of age and is currently associated with an autoimmune response and is a connective tissue disease. Temporal arteritis can produce headache symptoms with vascular headache characteristics, the headache is throbbing, often accompanied by facial redness and swelling, the headache is often located in the superficial part of the scalp and the temporal and periorbital parts, the nature of the headache is intermittent or persistent drilling and throbbing pain, and accompanied by burning pain or whacking disease that is not found in other vascular headaches, characterized by lying down or heavy at night, the pain is aggravated when chewing, compression of the anterior ear screen artery can cause Headache, and common ocular symptoms such as blackness, visual field defect, central dark spot, photophobia, blurred vision, etc., so it is necessary to make a distinction with migraine. (l) Nature of headache: Temporal arteritis pain is like pounding or burning, which is aggravated by chewing, lying down, and at night; migraine does not have the above characteristics. (2) Ocular symptoms: Ocular symptoms of temporal arteritis are mostly seen as diplopia and loss of vision, which mostly appear when the disease is aggravated; migraine is mostly an aura symptom, which can appear before, after and during the onset of pain. (3) Facial symptoms: claudication of the jaw or tongue and Raynaud’s phenomenon of the tongue are characteristic symptoms of temporal arteritis; migraine is rare. (4) Muscular symptoms: Rheumatic polymyopathy is the prodromal symptom and the most common clinical manifestation of temporal arteritis, mainly manifesting as wandering myalgia, mostly involving the neck, shoulder gluteal band joints, hip and hip joints, with pain not in the joints themselves but around the joints, first involving one side and then both sides, characterized by stiffness and fixation of the joints in the early morning; migraine does not have the above manifestations. (5) Laboratory tests: Temporal arteritis has increased blood sedimentation, increased alkaline phosphokinase, glutamic transaminase and prothrombin time, and small arterial inflammation including histiocytes, lymphocytes and giant cells can be seen in temporal artery biopsy; migraine has no such manifestations. (6) Treatment: Temporal arteritis is sensitive to glucocorticoids (commonly known as hormones), which are effective in relieving pain and preventing deterioration of vision; migraine is effective in the treatment of ergotamine. The manifestations of the disease are similar, but the treatment is very different. Therefore, making a correct diagnosis early is important to prevent visual impairment and joint disorders caused by temporal arteritis.