Most heart injuries are caused by the front foot being hit by heavy objects, driving disks, etc., or falling from a height, which violently shocks the heart. Direct or indirect violence violently pushes the heart between the foot bone and the spine, thus causing heart injury. Sudden acceleration or deceleration can also cause the dangling heart to collide with the sternum or spine and suffer injury. The right ventricle is most susceptible to contusion due to its close proximity to the sternum. It is most likely to be contused. The degree and extent of cardiac contusion can range from small pieces of epicardial or endocardial hemorrhage to large pieces of myocardial hemorrhage and necrosis. Causes of cardiac injury: 1, gender and age. Heart injury often occurs in older women, the incidence of women is four times that of men. Most occur in elderly patients over 60 years of age, heart injury foreign literature reports that most occur in elderly patients over 70-80 years of age, may be related to older ventricular wall myocardium is thinner and increased brittleness. 2. Hypertension. In the acute phase of myocardial infarction, blood pressure continues to rise above 20/12kPa (150/90mmHg) is easy to rupture, and the incidence of cardiac rupture is three times higher than that of those with normal blood pressure. 3. Cardiac injury almost never occurs in the myocardium with better collateral circulation. Because of the protective effect of the collateral circulation on the subepicardial myocardium, even if acute blockage of the coronary artery leads to acute myocardial infarction, it may be limited to the subendocardial myocardium, or abnormal Q waves may appear, and the R waves may only become smaller but not disappear. Due to the protection of the subepicardial myocardium, the heart form does not expand outward and prevents heart injury. 4. The injury often occurs in the first acute transmural myocardial infarction. The patient has no significant previous history of angina pectoris or heart failure. Sudden coronary artery thrombosis or severe coronary artery spasm without adequate collateral circulation often leads to penetrating transmural myocardial infarction. This type of initial myocardial infarction is prone to rupture because of the absence of usual myocardial ischemia, absence of old scar tissue as a scaffold, and better myocardial contraction in the non-infarcted area, which acts as a cutter for the myocardium in the necrotic area when the peripheral myocardium contracts.