Drug-induced hepatitis usually occurs 1 to 4 weeks after drug administration, and the specific manifestations are more or less the same as other hepatitis, including hepatocellular damage, fatigue, loss of appetite, nausea and vomiting, yellow urine, discomfort in the liver area, liver enlargement with pressure pain, elevated transaminases, and elevated eosinophils in the blood picture. In some cases, bile stasis is the main cause of bile stasis in the small intrahepatic bile ducts. Severe drug-related liver damage can also cause extensive hepatic necrosis, resulting in heavy hepatitis, severe jaundice, coagulation dysfunction and hepatic encephalopathy, and in some cases, upper gastrointestinal bleeding. If the diagnosis and treatment are not timely, it may be life-threatening. Hepatocellular necrosis, with life-saving treatment, may be replaced by extensive fibrous connective tissue, resulting in post-necrotic cirrhosis.