The relationship between renal disease and hypertension was first described by the British scholar Richard Bright in the early 19th century. 1898 Tigersteclt and Bergmann obtained a water-soluble pressor substance (called renin) from the kidney skin of a healthy rabbit and injected it into another healthy rabbit could lead to The existence of renal pressor substances was confirmed by the discovery of the renal pressor system by Page and Helmer and Braun-Menedz in 1940, which further elucidated the pathogenesis of renal hypertension. . In 1937, Bulter cured hypertension in a pediatric patient with pyelonephritis by removing a kidney, and in 1938, Leadbetter and Burkland cured hypertension in a patient with renal artery occlusion by removing a kidney. Since the 1950s, the development of percutaneous femoral artery puncture and arterial angiography, as well as advances in the measurement of fractional renal function and renin activity, have improved and perfected the diagnostic techniques for renal vascular hypertension, thus providing a basis for the diagnosis and treatment of renal vascular hypertension. The continuous introduction of new anti-hypertensive drugs and the continuous updating of vascular surgery techniques have opened up a broad prospect for the treatment of renal vascular hypertension.