Periumbilical discoloration is one of the signs of acute hemorrhagic necrotizing pancreatitis. Acute hemorrhagic necrotizing pancreatitis is a type of acute pancreatitis, which is caused by the continued development of acute edematous pancreatitis lesions. There is extensive necrosis of pancreatic alveoli, fat and blood vessels, edema of pancreatic tissue, increase in size, and extensive hemorrhagic necrosis. The retroperitoneal space is heavily hemorrhagic with exudate. The omentum and tissues of the lining are digested by the exuded pancreatic enzymes. This type of pancreatitis is severe and develops rapidly, with many complications and high mortality. How to check the discoloration around the umbilicus? 1. Symptoms Sudden onset of severe epigastric pain or high fever during the treatment of acute edematous pancreatitis, diffuse peritonitis, paralytic intestinal obstruction, epigastric mass, gastrointestinal bleeding, neuropsychiatric symptoms, and shock. 2. Signs: full abdominal distension, pressure pain and rebound pain, mobile turbid sounds, loss of bowel sounds, subcutaneous fat necrosis due to leakage of bloody exudate containing pancreatic enzymes through the retroperitoneal space to the subcutis in a few patients, bruising of the abdominal wall on both sides and discoloration around the umbilicus. 3. Ancillary tests ① Blood and urine amylase may be continuously elevated or not due to extensive necrosis of the pancreas. Blood calcium decreases, and its value <1.74mmol/L (7mg%) is poor prognosis. Blood glucose is elevated and glycosuria develops. Positive serum methemoglobin. (ii) Bloody turbidity of abdominal puncture fluid, elevated amylase and lipase. (iii) B-mode ultrasonography shows enlarged pancreas with sparse internal light dot reflection. CT shows diffusely enlarged pancreas with irregular shape, blurred margins and widened peripancreatic gap. It must be differentiated from acute gastroenteritis, acute perforation of ulcer disease, gallstone acute cholecystitis, myocardial infarction, acute intestinal obstruction, ischemic embolism of mesenteric vessels, etc. Pediatric acute pancreatitis is relatively rare, and the onset is related to the spillage of pancreatic fluid into the interstitial pancreatic vein and its surrounding tissues. It is now mostly thought to be related to viral infection, drugs, obstruction of the pancreatic secretory ducts, and certain systemic diseases or overeating. At least half of them are caused by mumps virus or blunt injuries to the upper abdomen, and there are still 30% of cases where the cause cannot be found. The data suggest that acute pancreatitis has a pathophysiological process of acute microcirculatory disorders. Acute pancreatitis five general after non-surgical efficacy of about 3 to 7 days, the symptoms disappear and gradually heal. Hemorrhagic, necrotic pancreatitis is serious, the course of the disease is longer, death can be due to shock, can also form a limited abscess, and can be followed by pseudocysts of the pancreas.