Arteriovenous ductus arteriosus is a common congenital heart disease. The arterial duct is a physiological blood flow channel between the fetal aorta and the left pulmonary artery that supplies blood and oxygen to the fetus, but after birth it is a discarded channel that needs to be closed. 85% of infants have a closed arterial duct as an arterial ligament within 2 months of birth, and those who do not close after that time have an unclosed arterial duct. Failure to close the arterial duct causes shunting of aortic blood flow to the pulmonary artery, increasing the blood volume of the pulmonary circulation and gradually developing left ventricular hypertrophy and right ventricular hypertrophy as the disease progresses. When the pulmonary artery pressure reaches or exceeds the aortic pressure, a bidirectional or right-to-left shunt of blood occurs and the patient develops cyanosis and pestle finger, known as Eisenmenger’s syndrome, which can lead to right heart failure and death. The prevalence of ductus arteriosus can reach 15-20%, with a twofold higher prevalence in women than in men. The clinical symptoms of an unclosed arterial catheter are not always present. Those with thin diameter catheters and small fractional flow often have no obvious symptoms. Those with thick diameters and large fractional flows are often complicated by congestive heart failure, manifested by irritability, shortness of breath, fatigue, excessive sweating, feeding difficulties, and poor growth. When the disease progresses to severe pulmonary hypertension with a right-to-left shunt, lower body cyanosis and pestle-like fingers (toes) appear. If a heart murmur is found, it is recommended to go to a regular hospital as soon as possible for a cardiac ultrasound to see if there is an arteriovenous ductus arteriosus.