Patients with cerebral infarction presenting with increased urine output may be due to medications. Patients with large cerebral infarction and brainstem infarction can have cerebral edema and increased intracranial pressure. Increased intracranial pressure is usually treated routinely with cranial pressure lowering therapy, and the main medications are mannitol, glyceryl fructose, and tachyphylaxis, which have certain cranial pressure-lowering effects, as well as diuretic effects, which may cause increased urination in patients. It may also be due to the patients with cerebral infarction combined with high blood pressure, certain blood pressure-lowering drugs may also cause increased urine output. In addition, it may be caused by the cerebral infarction itself, mainly seen in the paracentral lobe of the lesion next to the central lobe, the central lobe infarction, the patient can appear urinary and fecal dysfunction, manifested as urinary incontinence, urinary retention. Patients with urinary incontinence mainly have increased frequency of urination, which is not easy to control, and patients with urinary retention usually have difficulty urinating. If patients have urinary retention, they should be urinated down the urinary catheter in a timely manner to prevent excessive retention of urine, which can cause urinary tract infection and bladder rupture.