Reflux laryngitis is a disease that has been widely recognized by otorhinolaryngologists only in recent years, but in fact it is very common in clinical work. However, due to the lack of awareness in the past, the disease was misdiagnosed as common chronic laryngitis for a long time, and the lack of treatment for the cause of the disease led to no significant relief of symptoms, which troubled many patients for a long time and seriously reduced the quality of life of patients. Recent studies have found that the incidence of laryngopharyngeal reflux is very high in the population, accounting for 10% of all patients seen in ENT clinics and 50% of patients with hoarseness. As the name suggests, laryngopharyngeal reflux is caused by the reflux of stomach contents into the pharynx, which irritates and damages the pharyngeal mucosa and causes the corresponding symptoms. Causes: The refluxed acid directly irritates the laryngeal mucosa causing damage and complaints of discomfort. The normal protective substances in the laryngeal epithelium are missing in patients with laryngopharyngeal reflux, which together weaken the mucosal defense mechanism. At the same time, the pharyngeal mucosa lacks the motor contouring ability and salivary neutralization of the esophagus and is therefore significantly more sensitive to reflux stimuli than the latter. The refluxed material can stimulate the distal esophagus and cause a vagal reflex, which triggers a chronic cough and throat clearing that can cause damage to the vocal fold mucosa, and can cause a relaxation reflex of the upper esophageal sphincter, which allows the refluxed material to enter the pharynx and cause damage. Symptoms: pharyngeal foreign body sensation or hysterical ball sensation; hoarseness and dysphonia; chronic cough: mostly irritating dry cough; throat clearing, sore throat, dyspnea, halitosis, increased mucous secretions in the pharynx, dry throat, etc. Laryngoscopy: Patients with laryngopharyngeal reflux have some specific manifestations on laryngoscopy. Interarytenoid edema, pseudo-vocal fold grooves, edematous erythema in the posterior cricoid region, mucosal hypertrophy, vocal fold polyps and ulcers, shallow or absent laryngeal chambers, pebble-like changes in the pharynx, diffuse laryngitis, granulomas, subglottic stenosis, and stiffness of the cricoarytenoid joint are thought to be frequently seen in patients with laryngopharyngeal reflux. However, there is a lack of recognized specific microscopic manifestations that can be used to make a definitive diagnosis. Complications: Laryngopharyngeal reflux may be associated with laryngeal cancer, esophageal adenocarcinoma, secretory otitis media, laryngeal stenosis, laryngeal wheezing, chronic rhinosinusitis, obstructive sleep hypoventilation syndrome, dental enamel damage, and infant death syndrome. Acid suppression therapy combined with lifestyle changes: mainly includes avoiding eating before bedtime, elevating the head of the bed, reducing dinner intake, avoiding overeating, abstaining from smoking, alcohol, strong tea and coffee and high-fat foods, sweets, acidic fruits (oranges, prunes, etc.), and weight loss. Medication: Omeprazole twice daily before meals, together with macrocyclic lipids to promote gastrointestinal motility, and symptomatic throat medications (sweet orange and ice plum tablets, blue qin oral solution, etc.).