Patients will experience elevated blood pressure during the acute period of cerebral infarction, mainly as a stress response to an acute cerebrovascular event. Many patients with cerebral infarction have elevated blood pressure during the acute period because the local cerebral blood flow autoregulation dysfunction in the infarcted area is caused, and in order to make the blood flow in the ischemic semidark zone more balanced, so the body compensates by making the blood pressure rise. Do not actively lower the blood pressure during this period; maintaining a moderate cerebral perfusion pressure is beneficial to the recovery of the disease. If blood pressure is controlled too low, resulting in insufficient cerebral perfusion, the infarct area will expand and aggravate the patient’s condition. During the acute phase, the principle of blood pressure lowering is mainly to control the patient’s systolic blood pressure to within 25%, so that the patient’s high pressure is controlled at 150-160 mmHg and low pressure is 80-100 mmHg.