Cerebral atherosclerosis is also a risk factor for cerebrovascular disease. Cerebral atherosclerosis refers to the thickening of the elastic layer of the arterial wall, hardening of the wall, roughness of the intima and narrowing of the lumen. Its onset is related to hypertension, hyperlipidemia, diabetes and other factors, and it is a lipid metabolism disorder disease. Due to the disorder of lipid metabolism, lipids are deposited on the intima of arteries, forming atheromatous plaques, and these plaques protrude into the canal, narrowing or occluding the canal lumen. Cerebral blood vessels are prone to cerebral thrombosis due to lipid deposition, slow blood flow and increased blood viscosity. In addition, the cerebral vascular wall is vulnerable to the formation of micro aneurysms due to lipid accumulation, damage to the endothelium, and proliferation of connective tissue, and when the blood pressure rises suddenly, it is easy to rupture and bleed, and cerebral hemorrhage occurs. It can be seen that whether it is ischemic cerebrovascular disease or hemorrhagic cerebrovascular disease, it occurs on the basis of cerebral atherosclerosis, so cerebral atherosclerosis is also an important risk factor causing cerebrovascular disease. Smoking, alcohol consumption and mental factors: Smoking is also a very important risk factor for stroke, second only to age and hypertension, smoking can increase the plasma fibrin content, which can also cause cerebral hemangioma, etc. The risk of stroke is related to the number of cigarettes smoked, and some studies have shown that smoking can increase the incidence of subarachnoid hemorrhage due to aneurysm rupture, and the increase in smoking can further worsen hypertension and atherosclerosis . Smoking as an independent factor of stroke, timely cessation of smoking has a preventive effect on stroke, especially for hypertensive patients under 60 years of age, or patients with other comorbidities such as cardiomyopathy, diabetes mellitus and hyperlipidemia, it is more important to carry out practical and effective smoking cessation. The main effect of nicotine is to excite the sympathetic nerves, causing an increase in the release of adrenaline and norepinephrine, leading to an increase in blood pressure. Directly acting on the arterial wall to make it fatty and increase the adhesion and aggregation of platelets; heavy smoking can increase the content of plasma fibrinogen, increase blood viscosity and vascular wall damage, stimulate sympathetic nerves to make vasoconstriction, blood pressure, thereby reducing cerebral blood flow, promoting ischemic cerebrovascular disease. Long-term chronic alcohol consumption or alcoholism can cause hypertensive atherosclerosis, which can cause abnormal platelet function and activity. Long-term heavy drinking or alcoholism can increase platelet aggregation and blood flow coagulation, thereby reducing local cerebral blood flow; excessive drinking increases the risk of stroke, and alcoholism and hypertension synergistically cause a sudden rise in stroke. Therefore, smoking, alcohol consumption and mental health are also major contributing factors to ischemic cerebrovascular disease. Quit smoking, limit alcohol, and drink daily for men who are alcoholics.