The prevalence of G after febrile convulsions is 2% to 7%, which is 2 to 10 times higher than that of the normal population; 10% to 15% of patients with G have a history of FS. The probability of having at least one fever-free convulsion after FS ranges from 0.25% to 33% in different reports and is related to the length of follow-up. The main risk factors for secondary epileptic G in children with FS are still supported by the current study: (1) abnormal neurological development; (2) history of idiopathic or hereditary epileptic G in first-degree relatives; (3) CFS. The probability of developing epileptic G at age 7 years is 1% for those without these risk factors, 2% for those with one risk factor, and 10% for those with two or three risk factors. In addition, the short duration of fever before the seizure and the high number of FS seizures are also risk factors for secondary epileptic G. Some epileptic G syndromes with FS onset may present with FS in the early stages and gradually show “unusual” clinical and EEG manifestations as the disease progresses, which requires attention. A retrospective analysis showed that G syndromes with a previous history of FS include: refractory medial temporal lobe G (80%), temporal lobe G (25%), childhood akathisia G (20%), idiopathic benign occipital lobe G in children (17%), myoclonic-standing inability to seize G (11%-28%), juvenile myoclonic G (5%-10%), Lennox- Dravet’s syndrome and generalized epileptic G with febrile convulsion add-on (GEFS+) also have a definite correlation with FS. In particular, it should be noted that some epileptic G syndromes, which can present early as FS, gradually present other specific clinical features that should be taken into account in the diagnosis.