Ischemic optic neuropathy is most often seen in the elderly, over 60 years of age, with sequential onset in one or both eyes. It is important to treat this disease promptly when it occurs. The disease is caused by circulatory disorders of the small vessels that nourish the anterior segment of the optic nerve, occlusion of the regressive branch of the short posterior ciliary artery; or involvement of the soft meningeal vessels of the optic nerve, which causes insufficient blood supply to the optic papilla and acute ischemia and hypoxia and edema; low or high intraocular pressure, which makes the perfusion pressure of the small vessels of the optic papilla out of balance with the intraocular pressure, can also cause optic papillary edema. The clinical manifestations of this disease are mainly sudden changes in visual acuity and visual field. Visual acuity is suddenly blurred and decreases to the point of blindness. The visual field is initially a fan-shaped defect below the nose, which later expands into eccentricity or centripetal narrowing, sometimes connected to a physiological blind spot. Early on, one quadrant of the optic papilla is lightly edematous, which may gradually extend to half or the entire optic papilla with a small amount of exudate and hemorrhagic spots. Within a few weeks, the optic papilla edema may resolve on its own, but primary optic nerve atrophy will soon occur. Treatment of ischemic optic neuropathy: postbulbar injection or intravenous corticosteroid drip to reduce the edema and exudation of the optic nerve due to impaired blood circulation, and gradually reduce the dose to discontinue the drug after the edema subsides. Oral acetazolamide is used to reduce intraocular pressure, improve intravascular perfusion pressure of the optic nerve, and correct the disproportion between perfusion pressure and intraocular pressure. Apply large amounts of B vitamins and vasodilators to enhance optic nerve nutrition.