Stroke Cerebrovascular disease

  1, emergency treatment of cerebrovascular disease in the acute phase A, B, C measures A (airway) and B (breathing): especially for patients in severe coma, ball palsy, convulsions, to ensure the usual airway and oxygen supply. C(circulation): pay attention to cranial hypertension, cerebral herniation, cardiogenic, medical (massive dehydration) resulting in circulatory disorders.  2, blood pressure problems in acute cerebrovascular disease: The survey found that systolic blood pressure higher than 160 mmHg reached 35% and diastolic blood pressure higher than 90 mmHg reached 52% in the acute phase of acute cerebrovascular disease. Therefore, the increase in blood pressure is a more common problem. Managing blood pressure also appears to be more important. High blood pressure can theoretically ensure the blood supply to the brain, but it cannot be denied that it can lead to vasospasm and affect the function of vital organs.  Acute cerebrovascular disease with a systolic blood pressure no higher than 220 mmHg and a diastolic blood pressure no higher than 110 mmHg may not require treatment. However: for non-cranial hypertensive blood pressure increase, as long as the blood pressure is not lowered too quickly and excessively, slowly stabilizing the blood pressure to near normal is helpful for treatment.  3.Blood volume expansion and blood thinning treatment: low molecular dextrose, 706 plasma substitute and other treatments.  4.Thermolytic treatment: thrombin inhibitors: heparin and heparin-like. Low-molecular heparin is safe, efficacious and reliable, and even foreign reports have shown no serious adverse consequences in patients with hemorrhagic infarction. Fibrinolytic agents: tissue-type fibrinogen activator (tPA), urokinase, streptokinase, snake venom, earthworm kinase, etc. Currently, snake venom is more frequently used, and its fibrin-lowering effect is better, but the indications for treatment should be strictly followed: within 6 hours of onset and without bleeding tendency (note that more than 1/3 of patients with cerebral embolism will have bleeding from the infarct site).